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10.3390/biomedicines10061332

http://scihub22266oqcxt.onion/10.3390/biomedicines10061332
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35740354!9220124!35740354
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suck abstract from ncbi


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pmid35740354      Biomedicines 2022 ; 10 (6): ä
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  • Unbalanced IDO1/IDO2 Endothelial Expression and Skewed Keynurenine Pathway in the Pathogenesis of COVID-19 and Post-COVID-19 Pneumonia #MMPMID35740354
  • Chilosi M; Doglioni C; Ravaglia C; Martignoni G; Salvagno GL; Pizzolo G; Bronte V; Poletti V
  • Biomedicines 2022[Jun]; 10 (6): ä PMID35740354show ga
  • Despite intense investigation, the pathogenesis of COVID-19 and the newly defined long COVID-19 syndrome are not fully understood. Increasing evidence has been provided of metabolic alterations characterizing this group of disorders, with particular relevance of an activated tryptophan/kynurenine pathway as described in this review. Recent histological studies have documented that, in COVID-19 patients, indoleamine 2,3-dioxygenase (IDO) enzymes are differentially expressed in the pulmonary blood vessels, i.e., IDO1 prevails in early/mild pneumonia and in lung tissues from patients suffering from long COVID-19, whereas IDO2 is predominant in severe/fatal cases. We hypothesize that IDO1 is necessary for a correct control of the vascular tone of pulmonary vessels, and its deficiency in COVID-19 might be related to the syndrome's evolution toward vascular dysfunction. The complexity of this scenario is discussed in light of possible therapeutic manipulations of the tryptophan/kynurenine pathway in COVID-19 and post-acute COVID-19 syndromes.
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