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10.3389/fimmu.2022.903498 http://scihub22266oqcxt.onion/10.3389/fimmu.2022.903498 35711451!9196331!35711451     free     free     free Deprecated: Implicit conversion from float 235.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 235.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 235.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Front+Immunol 2022 ; 13 (ä): 903498 Nephropedia Template TP {{tp|p=35711451|t=2022. Autophagy Hijacking in PBMC From COVID-19 Patients Results in Lymphopenia |pdf=|usr=}}{{35711451}} gab.com Text Autophagy Hijacking in PBMC From COVID-19 Patients Results in Lymphopenia JO: Front Immunol http://www.kidney.de/pget.php?&tw=1&pmid=35711451 #FOAvip Autophagy is a homeostatic process responsible for the self-digestion of intracellular components and antimicrobial defense by inducing the degradation of pathogens into autophagolysosomes. Recent findings suggest an involvement of this process in severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection. However, the role of autophagy in the immunological mechanisms of coronavirus disease 2019 (COVID-19) pathogenesis remains largely unexplored. This study reveals the presence of autophagy defects in peripheral immune cells from COVID-19 patients. The impairment of the autophagy process resulted in a higher percentage of lymphocytes undergoing apoptosis in COVID-19 patients. Moreover, the inverse correlation between autophagy markers levels and peripheral lymphocyte counts in COVID-19 patients confirms how a defect in autophagy might contribute to lymphopenia, causing a reduction in the activation of viral defense. These results provided intriguing data that could help in understanding the cellular underlying mechanisms in COVID-19 infection, especially in severe forms. Twit Text FOAVip Autophagy Hijacking in PBMC From COVID-19 Patients Results in Lymphopenia ????Front Immunol http://www.kidney.de/pget.php?&tw=1&pmid=35711451 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=35711451 #FOAvip English Wikipedia <ref>{{Cite pmid|35711451}}</ref> Autophagy Hijacking in PBMC From COVID-19 Patients Results in Lymphopenia #MMPMID35711451 Barbati C; Celia AI; Colasanti T; Vomero M; Speziali M; Putro E; Buoncuore G; Savino F; Colafrancesco S; Ucci FM; Ciancarella C; Balbinot E; Scarpa S; Natalucci F; Pellegrino G; Ceccarelli F; Spinelli FR; Mastroianni CM; Conti F; Alessandri C Front Immunol 2022[]; 13 (ä): 903498 PMID35711451show ga Autophagy is a homeostatic process responsible for the self-digestion of intracellular components and antimicrobial defense by inducing the degradation of pathogens into autophagolysosomes. Recent findings suggest an involvement of this process in severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection. However, the role of autophagy in the immunological mechanisms of coronavirus disease 2019 (COVID-19) pathogenesis remains largely unexplored. This study reveals the presence of autophagy defects in peripheral immune cells from COVID-19 patients. The impairment of the autophagy process resulted in a higher percentage of lymphocytes undergoing apoptosis in COVID-19 patients. Moreover, the inverse correlation between autophagy markers levels and peripheral lymphocyte counts in COVID-19 patients confirms how a defect in autophagy might contribute to lymphopenia, causing a reduction in the activation of viral defense. These results provided intriguing data that could help in understanding the cellular underlying mechanisms in COVID-19 infection, especially in severe forms. |*COVID-19[MESH] |*Lymphopenia[MESH] |Autophagy[MESH] |Humans[MESH] |Leukocytes, Mononuclear[MESH]Autophagy Hijacking in PBMC From COVID-19 Patients Results in Lymphope(epmc).pdf DeepDyve Pubget Overpricing