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10.1016/bs.ircmb.2022.01.001 http://scihub22266oqcxt.onion/10.1016/bs.ircmb.2022.01.001 35461655!8968207!35461655     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=35461655&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Int+Rev+Cell+Mol+Biol 2022 ; 367 (ä): 1-28 Nephropedia Template TP {{tp|p=35461655|t=2022. Pulmonary macrophages and SARS-Cov2 infection |pdf=|usr=}}{{35461655}} gab.com Text Pulmonary macrophages and SARS-Cov2 infection JO: Int Rev Cell Mol Biol http://www.kidney.de/pget.php?&tw=1&pmid=35461655 #FOAvip The emergence of severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) has led to the largest global pandemic in living memory, with between 4.5 and 15M deaths globally from coronavirus disease 2019 (COVID-19). This has led to an unparalleled global, collaborative effort to understand the pathogenesis of this devastating disease using state-of-the-art technologies. A consistent feature of severe COVID-19 is dysregulation of pulmonary macrophages, cells that under normal physiological conditions play vital roles in maintaining lung homeostasis and immunity. In this article, we will discuss a selection of the pivotal findings examining the role of monocytes and macrophages in SARS-CoV-2 infection and place this in context of recent advances made in understanding the fundamental immunobiology of these cells to try to understand how key homeostatic cells come to be a central pathogenic component of severe COVID-19 and key cells to target for therapeutic gain. Twit Text FOAVip Pulmonary macrophages and SARS-Cov2 infection ????Int Rev Cell Mol Biol http://www.kidney.de/pget.php?&tw=1&pmid=35461655 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=35461655 #FOAvip English Wikipedia <ref>{{Cite pmid|35461655}}</ref> Pulmonary macrophages and SARS-Cov2 infection #MMPMID35461655 Bain CC; Lucas CD; Rossi AG Int Rev Cell Mol Biol 2022[]; 367 (ä): 1-28 PMID35461655show ga The emergence of severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) has led to the largest global pandemic in living memory, with between 4.5 and 15M deaths globally from coronavirus disease 2019 (COVID-19). This has led to an unparalleled global, collaborative effort to understand the pathogenesis of this devastating disease using state-of-the-art technologies. A consistent feature of severe COVID-19 is dysregulation of pulmonary macrophages, cells that under normal physiological conditions play vital roles in maintaining lung homeostasis and immunity. In this article, we will discuss a selection of the pivotal findings examining the role of monocytes and macrophages in SARS-CoV-2 infection and place this in context of recent advances made in understanding the fundamental immunobiology of these cells to try to understand how key homeostatic cells come to be a central pathogenic component of severe COVID-19 and key cells to target for therapeutic gain. |*COVID-19[MESH] |Humans[MESH] |Macrophages[MESH] |Macrophages, Alveolar[MESH] |RNA, Viral[MESH]Pulmonary macrophages and SARS-Cov2 infection (epmc).pdf DeepDyve Pubget Overpricing