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10.3390/ijms23073868 http://scihub22266oqcxt.onion/10.3390/ijms23073868 35409226!8998935!35409226     free     free     free Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Int+J+Mol+Sci 2022 ; 23 (7): ä Nephropedia Template TP {{tp|p=35409226|t=2022. Beyond Hemostasis: Platelet Innate Immune Interactions and Thromboinflammation |pdf=|usr=}}{{35409226}} gab.com Text Beyond Hemostasis: Platelet Innate Immune Interactions and Thromboinflammation JO: Int J Mol Sci http://www.kidney.de/pget.php?&tw=1&pmid=35409226 #FOAvip There is accumulating evidence that platelets play roles beyond their traditional functions in thrombosis and hemostasis, e.g., in inflammatory processes, infection and cancer, and that they interact, stimulate and regulate cells of the innate immune system such as neutrophils, monocytes and macrophages. In this review, we will focus on platelet activation in hemostatic and inflammatory processes, as well as platelet interactions with neutrophils and monocytes/macrophages. We take a closer look at the contributions of major platelet receptors GPIb, alpha(IIb)beta(3), TLT-1, CLEC-2 and Toll-like receptors (TLRs) as well as secretions from platelet granules on platelet-neutrophil aggregate and neutrophil extracellular trap (NET) formation in atherosclerosis, transfusion-related acute lung injury (TRALI) and COVID-19. Further, we will address platelet-monocyte and macrophage interactions during cancer metastasis, infection, sepsis and platelet clearance. Twit Text FOAVip Beyond Hemostasis: Platelet Innate Immune Interactions and Thromboinflammation ????Int J Mol Sci http://www.kidney.de/pget.php?&tw=1&pmid=35409226 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=35409226 #FOAvip English Wikipedia <ref>{{Cite pmid|35409226}}</ref> Beyond Hemostasis: Platelet Innate Immune Interactions and Thromboinflammation #MMPMID35409226 Mandel J; Casari M; Stepanyan M; Martyanov A; Deppermann C Int J Mol Sci 2022[Mar]; 23 (7): ä PMID35409226show ga There is accumulating evidence that platelets play roles beyond their traditional functions in thrombosis and hemostasis, e.g., in inflammatory processes, infection and cancer, and that they interact, stimulate and regulate cells of the innate immune system such as neutrophils, monocytes and macrophages. In this review, we will focus on platelet activation in hemostatic and inflammatory processes, as well as platelet interactions with neutrophils and monocytes/macrophages. We take a closer look at the contributions of major platelet receptors GPIb, alpha(IIb)beta(3), TLT-1, CLEC-2 and Toll-like receptors (TLRs) as well as secretions from platelet granules on platelet-neutrophil aggregate and neutrophil extracellular trap (NET) formation in atherosclerosis, transfusion-related acute lung injury (TRALI) and COVID-19. Further, we will address platelet-monocyte and macrophage interactions during cancer metastasis, infection, sepsis and platelet clearance. |*COVID-19[MESH] |*Thrombosis/pathology[MESH] |Blood Platelets/pathology[MESH] |Hemostasis[MESH] |Humans[MESH] |Immunity, Innate[MESH] |Inflammation/pathology[MESH] |Neutrophils/pathology[MESH] |Platelet Activation[MESH]Beyond Hemostasis: Platelet Innate Immune Interactions and Thromboinfl(epmc).pdf DeepDyve Pubget Overpricing