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10.1042/CS20220028 http://scihub22266oqcxt.onion/10.1042/CS20220028 35348182!8965847!35348182     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Clin+Sci+(Lond) 2022 ; 136 (6): 431-434 Nephropedia Template TP {{tp|p=35348182|t=2022. SARS-CoV-2 spike protein causes cardiovascular disease independent of viral infection |pdf=|usr=}}{{35348182}} gab.com Text SARS-CoV-2 spike protein causes cardiovascular disease independent of viral infection JO: Clin Sci (Lond) http://www.kidney.de/pget.php?&tw=1&pmid=35348182 #FOAvip The SARS-CoV-2 virus that results in COVID-19 has been found to damage multiple organs beyond the lung. Interestingly, the SARS-CoV-2 spike (S) protein can be found circulating in the blood of COVID-19 patients. Experimental findings are demonstrating that the circulating S protein can bind to receptors resulting in inflammation and cell, tissue, and organ damage. Avolio et al. previously determined that the S protein acting through the cluster of differentiation 147 (CD147) receptor, and another unknown mechanism had detrimental effects on human cardiac pericytes (Clin Sci (Lond) (2021) 135 (24): 2667-2689. DOI: 10.1042/CS20210735). These findings support the notion that circulating SARS-CoV-2 S protein could contribute to cardiovascular disease independent of viral infection. Future studies are needed to determine the effect of the S protein on pericytes in other organs and evaluate the effectiveness of CD147 receptor-blocking therapies to decrease organ damage caused by the S protein. Twit Text FOAVip SARS-CoV-2 spike protein causes cardiovascular disease independent of viral infection ????Clin Sci (Lond) http://www.kidney.de/pget.php?&tw=1&pmid=35348182 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=35348182 #FOAvip English Wikipedia <ref>{{Cite pmid|35348182}}</ref> SARS-CoV-2 spike protein causes cardiovascular disease independent of viral infection #MMPMID35348182 Imig JD Clin Sci (Lond) 2022[Mar]; 136 (6): 431-434 PMID35348182show ga The SARS-CoV-2 virus that results in COVID-19 has been found to damage multiple organs beyond the lung. Interestingly, the SARS-CoV-2 spike (S) protein can be found circulating in the blood of COVID-19 patients. Experimental findings are demonstrating that the circulating S protein can bind to receptors resulting in inflammation and cell, tissue, and organ damage. Avolio et al. previously determined that the S protein acting through the cluster of differentiation 147 (CD147) receptor, and another unknown mechanism had detrimental effects on human cardiac pericytes (Clin Sci (Lond) (2021) 135 (24): 2667-2689. DOI: 10.1042/CS20210735). These findings support the notion that circulating SARS-CoV-2 S protein could contribute to cardiovascular disease independent of viral infection. Future studies are needed to determine the effect of the S protein on pericytes in other organs and evaluate the effectiveness of CD147 receptor-blocking therapies to decrease organ damage caused by the S protein. |*COVID-19[MESH] |*Cardiovascular Diseases[MESH] |*Virus Diseases[MESH] |Humans[MESH] |SARS-CoV-2[MESH]SARS-CoV-2 spike protein causes cardiovascular disease independent of (epmc).pdf DeepDyve Pubget Overpricing