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10.1016/j.yjmcc.2022.03.007

http://scihub22266oqcxt.onion/10.1016/j.yjmcc.2022.03.007
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suck abstract from ncbi


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pmid35339512      J+Mol+Cell+Cardiol 2022 ; 167 (ä): 92-96
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  • Endothelin-1 is increased in the plasma of patients hospitalised with Covid-19 #MMPMID35339512
  • Abraham GR; Kuc RE; Althage M; Greasley PJ; Ambery P; Maguire JJ; Wilkinson IB; Hoole SP; Cheriyan J; Davenport AP
  • J Mol Cell Cardiol 2022[Jun]; 167 (ä): 92-96 PMID35339512show ga
  • Virus induced endothelial dysregulation is a well-recognised feature of severe Covid-19 infection. Endothelin-1 (ET-1) is the most highly expressed peptide in endothelial cells and a potent vasoconstrictor, thus representing a potential therapeutic target. ET-1 plasma levels were measured in a cohort of 194 Covid-19 patients stratified according to the clinical severity of their illness. Hospitalised patients, including those who died and those developing acute myocardial or kidney injury, had significantly elevated ET-1 plasma levels during the acute phase of infection. The results support the hypothesis that endothelin receptor antagonists may provide clinical benefit for certain Covid-19 patients.
  • |*COVID-19[MESH]
  • |*Endothelin-1[MESH]
  • |Endothelial Cells[MESH]
  • |Endothelin Receptor Antagonists[MESH]
  • |Humans[MESH]
  • |Receptor, Endothelin A[MESH]
  • |Receptors, Endothelin[MESH]


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