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10.3390/ijms23063346 http://scihub22266oqcxt.onion/10.3390/ijms23063346 35328769!8954705!35328769     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=35328769&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Int+J+Mol+Sci 2022 ; 23 (6): ? Nephropedia Template TP {{tp|p=35328769|t=2022. Pathophysiology of Atherosclerosis |pdf=|usr=}}{{35328769}} gab.com Text Pathophysiology of Atherosclerosis JO: Int J Mol Sci http://www.kidney.de/pget.php?&tw=1&pmid=35328769 #FOAvip Atherosclerosis is the main risk factor for cardiovascular disease (CVD), which is the leading cause of mortality worldwide. Atherosclerosis is initiated by endothelium activation and, followed by a cascade of events (accumulation of lipids, fibrous elements, and calcification), triggers the vessel narrowing and activation of inflammatory pathways. The resultant atheroma plaque, along with these processes, results in cardiovascular complications. This review focuses on the different stages of atherosclerosis development, ranging from endothelial dysfunction to plaque rupture. In addition, the post-transcriptional regulation and modulation of atheroma plaque by microRNAs and lncRNAs, the role of microbiota, and the importance of sex as a crucial risk factor in atherosclerosis are covered here in order to provide a global view of the disease. Twit Text FOAVip Pathophysiology of Atherosclerosis ????Int J Mol Sci http://www.kidney.de/pget.php?&tw=1&pmid=35328769 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=35328769 #FOAvip English Wikipedia <ref>{{Cite pmid|35328769}}</ref> Pathophysiology of Atherosclerosis #MMPMID35328769 Jebari-Benslaiman S; Galicia-Garcia U; Larrea-Sebal A; Olaetxea JR; Alloza I; Vandenbroeck K; Benito-Vicente A; Martin C Int J Mol Sci 2022[Mar]; 23 (6): ? PMID35328769show ga Atherosclerosis is the main risk factor for cardiovascular disease (CVD), which is the leading cause of mortality worldwide. Atherosclerosis is initiated by endothelium activation and, followed by a cascade of events (accumulation of lipids, fibrous elements, and calcification), triggers the vessel narrowing and activation of inflammatory pathways. The resultant atheroma plaque, along with these processes, results in cardiovascular complications. This review focuses on the different stages of atherosclerosis development, ranging from endothelial dysfunction to plaque rupture. In addition, the post-transcriptional regulation and modulation of atheroma plaque by microRNAs and lncRNAs, the role of microbiota, and the importance of sex as a crucial risk factor in atherosclerosis are covered here in order to provide a global view of the disease. |*Atherosclerosis/metabolism[MESH] |*Calcinosis/complications[MESH] |*Cardiovascular Diseases/metabolism[MESH] |*Plaque, Atherosclerotic[MESH] |Humans[MESH]Pathophysiology of Atherosclerosis (epmc).pdf DeepDyve Pubget Overpricing