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10.1039/d2mo00055e

http://scihub22266oqcxt.onion/10.1039/d2mo00055e
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35284913!ä!35284913

suck abstract from ncbi


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pmid35284913      Mol+Omics 2022 ; 18 (5): 408-416
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  • Th1, Th2 and Th17 inflammatory pathways synergistically predict cardiometabolic protein expression in serum of COVID-19 patients #MMPMID35284913
  • Michels JR; Nazrul MS; Adhikari S; Wilkins D; Pavel AB
  • Mol Omics 2022[Jun]; 18 (5): 408-416 PMID35284913show ga
  • A predominant source of complication in SARS-CoV-2 patients arises from a severe systemic inflammation that can lead to tissue damage and organ failure. The high inflammatory burden of this viral infection often results in cardiovascular comorbidities. A better understanding of the interaction between immune pathways and cardiovascular proteins might inform medical decisions and therapeutic approaches. In this study we hypothesized that helper T-cell inflammatory pathways (Th1, Th2 and Th17) synergistically correlate with cardiometabolic proteins in serum of COVID-19 patients. We found that Th1, Th2 and Th17 cytokines and chemokines are able to predict expression of 186 cardiometabolic proteins profiled by Olink proteomics.
  • |*COVID-19[MESH]
  • |*Cardiovascular Diseases/metabolism[MESH]
  • |Humans[MESH]
  • |Proteomics[MESH]
  • |SARS-CoV-2[MESH]
  • |Th1 Cells/metabolism[MESH]
  • |Th17 Cells/metabolism[MESH]


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