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10.3390/antiox11020386 http://scihub22266oqcxt.onion/10.3390/antiox11020386 35204269!8869563!35204269     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=35204269&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Antioxidants+(Basel) 2022 ; 11 (2): ? Nephropedia Template TP {{tp|p=35204269|t=2022. The Beneficial Effect of Carvacrol in HL-1 Cardiomyocytes Treated with LPS-G: Anti-Inflammatory Pathway Investigations |pdf=|usr=}}{{35204269}} gab.com Text The Beneficial Effect of Carvacrol in HL-1 Cardiomyocytes Treated with LPS-G: Anti-Inflammatory Pathway Investigations JO: Antioxidants (Basel) http://www.kidney.de/pget.php?&tw=1&pmid=35204269 #FOAvip Carvacrol (CAR), a natural phenolic monoterpene, possesses different biological activities, such as anti-inflammatory and antioxidant activities. The current study aimed to evaluate the response of HL-1 cardiomyocytes to an inflammatory stimulus triggered by lipopolysaccharide from Porphyromonas gingivalis (LPS-G), alone or in co-treatment with CAR, to investigate the potential protective role of CAR in the inflammatory process through modulation of the TLR4/NFkappaB/NALP3/IL-1beta pathway and ROS production. In an in vitro experiment, HL-1 cardiomyocytes were exposed to LPS-G and incubated with CAR. We evaluated the anti-inflammatory effect of CAR by the reduction in TLR4, NFkappaB, NALP3, and IL-1beta expression using immunofluorescence staining. Western blot analysis also validated the modulation of the TLR4/NFkappaB/NALP3/IL-1beta pathway. ROS analyses confirmed the protective effects of CAR. Our results suggest that CAR could provide a significant protection role against inflammatory stimulus generated by LPS-G, involving the suppression of the TLR4/NFkappaB/NALP3/IL-1beta signaling pathway. Twit Text FOAVip The Beneficial Effect of Carvacrol in HL-1 Cardiomyocytes Treated with LPS-G: Anti-Inflammatory Pathway Investigations ????Antioxidants (Basel) http://www.kidney.de/pget.php?&tw=1&pmid=35204269 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=35204269 #FOAvip English Wikipedia <ref>{{Cite pmid|35204269}}</ref> The Beneficial Effect of Carvacrol in HL-1 Cardiomyocytes Treated with LPS-G: Anti-Inflammatory Pathway Investigations #MMPMID35204269 Marconi GD; Della Rocca Y; Fonticoli L; Guarnieri S; Carradori S; Rajan TS; Pizzicannella J; Diomede F Antioxidants (Basel) 2022[Feb]; 11 (2): ? PMID35204269show ga Carvacrol (CAR), a natural phenolic monoterpene, possesses different biological activities, such as anti-inflammatory and antioxidant activities. The current study aimed to evaluate the response of HL-1 cardiomyocytes to an inflammatory stimulus triggered by lipopolysaccharide from Porphyromonas gingivalis (LPS-G), alone or in co-treatment with CAR, to investigate the potential protective role of CAR in the inflammatory process through modulation of the TLR4/NFkappaB/NALP3/IL-1beta pathway and ROS production. In an in vitro experiment, HL-1 cardiomyocytes were exposed to LPS-G and incubated with CAR. We evaluated the anti-inflammatory effect of CAR by the reduction in TLR4, NFkappaB, NALP3, and IL-1beta expression using immunofluorescence staining. Western blot analysis also validated the modulation of the TLR4/NFkappaB/NALP3/IL-1beta pathway. ROS analyses confirmed the protective effects of CAR. Our results suggest that CAR could provide a significant protection role against inflammatory stimulus generated by LPS-G, involving the suppression of the TLR4/NFkappaB/NALP3/IL-1beta signaling pathway. ?The Beneficial Effect of Carvacrol in HL-1 Cardiomyocytes Treated with(epmc).pdf DeepDyve Pubget Overpricing