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10.1016/j.jacbts.2021.12.003 http://scihub22266oqcxt.onion/10.1016/j.jacbts.2021.12.003 35194565!8849181!35194565     free     free     free Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\35194565.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       JACC+Basic+Transl+Sci 2022 ; 7 (3): 193-204 Nephropedia Template TP {{tp|p=35194565|t=2022. Erythrocytes Induce Vascular Dysfunction in COVID-19 |pdf=|usr=}}{{35194565}} gab.com Text Erythrocytes Induce Vascular Dysfunction in COVID-19 JO: JACC Basic Transl Sci http://www.kidney.de/pget.php?&tw=1&pmid=35194565 #FOAvip Current knowledge regarding mechanisms underlying cardiovascular complications in patients with COVID-19 is limited and urgently needed. We shed light on a previously unrecognized mechanism and unravel a key role of red blood cells, driving vascular dysfunction in patients with COVID-19 infection. We establish the presence of profound and persistent endothelial dysfunction in vivo in patients with COVID-19. Mechanistically, we show that targeting reactive oxygen species or arginase 1 improves vascular dysfunction mediated by red blood cells. These translational observations hold promise that restoring the redox balance in red blood cells might alleviate the clinical complications of COVID-19-associated vascular dysfunction. Twit Text FOAVip Erythrocytes Induce Vascular Dysfunction in COVID-19 ????JACC Basic Transl Sci http://www.kidney.de/pget.php?&tw=1&pmid=35194565 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=35194565 #FOAvip English Wikipedia <ref>{{Cite pmid|35194565}}</ref> Erythrocytes Induce Vascular Dysfunction in COVID-19 #MMPMID35194565 Mahdi A; Collado A; Tengbom J; Jiao T; Wodaje T; Johansson N; Farnebo F; Farnert A; Yang J; Lundberg JO; Zhou Z; Pernow J JACC Basic Transl Sci 2022[Mar]; 7 (3): 193-204 PMID35194565show ga Current knowledge regarding mechanisms underlying cardiovascular complications in patients with COVID-19 is limited and urgently needed. We shed light on a previously unrecognized mechanism and unravel a key role of red blood cells, driving vascular dysfunction in patients with COVID-19 infection. We establish the presence of profound and persistent endothelial dysfunction in vivo in patients with COVID-19. Mechanistically, we show that targeting reactive oxygen species or arginase 1 improves vascular dysfunction mediated by red blood cells. These translational observations hold promise that restoring the redox balance in red blood cells might alleviate the clinical complications of COVID-19-associated vascular dysfunction. äErythrocytes Induce Vascular Dysfunction in COVID-19 (epmc).pdf DeepDyve Pubget Overpricing