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10.4081/monaldi.2022.2213 http://scihub22266oqcxt.onion/10.4081/monaldi.2022.2213 35179012!ä!35179012 Deprecated: Implicit conversion from float 235.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 235.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Monaldi+Arch+Chest+Dis 2022 ; 92 (4): ä Nephropedia Template TP {{tp|p=35179012|t=2022. Endothelial dysfunction in COVID-19 patients assessed with Endo-PAT2000 |pdf=|usr=}}{{35179012}} gab.com Text Endothelial dysfunction in COVID-19 patients assessed with Endo-PAT2000 JO: Monaldi Arch Chest Dis http://www.kidney.de/pget.php?&tw=1&pmid=35179012 #FOAvip It has been widely reported that the Severe Acute Respiratory Syndrome Coronavirus 2 (SARS-CoV-2) attaches human cells by using the Angiotensin Converting Enzyme 2 (ACE2) receptor, but vascular impairment described during coronavirus disease 2019 (COVID-19) infection is primarily due to the direct involvement of the endothelial cells by the virus or secondarily to the inflammatory host response is currently unknown. We therefore aimed to demonstrate in vivo the presence of endothelial dysfunction in six COVID-19 patients without cardiovascular risk factors or pre-existing cardiac condition, using the Endo-PAT 2000, a device able to measure endothelial vasodilation function in a rapid and non-invasive way. Four patients were positive for endothelial dysfunction, with RHI values between 1.13-1.56 (average value 1.32, normal values >1.67); in one of the two negative patients the reported RHI value was slightly above the cutoff (1.72). Our findings confirm that COVID-19 patients are at higher risk of developing endothelial dysfunction. In addition, our results demonstrate that endothelial impairment may occur even in the absence of cardiovascular risk factors. Twit Text FOAVip Endothelial dysfunction in COVID-19 patients assessed with Endo-PAT2000 ????Monaldi Arch Chest Dis http://www.kidney.de/pget.php?&tw=1&pmid=35179012 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=35179012 #FOAvip English Wikipedia <ref>{{Cite pmid|35179012}}</ref> Endothelial dysfunction in COVID-19 patients assessed with Endo-PAT2000 #MMPMID35179012 Cimino G; Vizzardi E; Calvi E; Pancaldi E; Pascariello G; Bernardi N; Cersosimo A; Amore L; Inciardi RM; Raddino R; Metra M Monaldi Arch Chest Dis 2022[Feb]; 92 (4): ä PMID35179012show ga It has been widely reported that the Severe Acute Respiratory Syndrome Coronavirus 2 (SARS-CoV-2) attaches human cells by using the Angiotensin Converting Enzyme 2 (ACE2) receptor, but vascular impairment described during coronavirus disease 2019 (COVID-19) infection is primarily due to the direct involvement of the endothelial cells by the virus or secondarily to the inflammatory host response is currently unknown. We therefore aimed to demonstrate in vivo the presence of endothelial dysfunction in six COVID-19 patients without cardiovascular risk factors or pre-existing cardiac condition, using the Endo-PAT 2000, a device able to measure endothelial vasodilation function in a rapid and non-invasive way. Four patients were positive for endothelial dysfunction, with RHI values between 1.13-1.56 (average value 1.32, normal values >1.67); in one of the two negative patients the reported RHI value was slightly above the cutoff (1.72). Our findings confirm that COVID-19 patients are at higher risk of developing endothelial dysfunction. In addition, our results demonstrate that endothelial impairment may occur even in the absence of cardiovascular risk factors. |*COVID-19[MESH] |*Vascular Diseases[MESH] |Angiotensin-Converting Enzyme 2[MESH] |Endothelial Cells[MESH] |Humans[MESH] |Peptidyl-Dipeptidase A[MESH]Endothelial dysfunction in COVID-19 patients assessed with Endo-PAT200(epmc).pdf DeepDyve Pubget Overpricing