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10.1016/j.biopha.2022.112718 http://scihub22266oqcxt.onion/10.1016/j.biopha.2022.112718 35176710!8841219!35176710     free     free     free Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Biomed+Pharmacother 2022 ; 148 (ä): 112718 Nephropedia Template TP {{tp|p=35176710|t=2022. Role of the renin-angiotensin system in NETosis in the coronavirus disease 2019 (COVID-19) |pdf=|usr=}}{{35176710}} gab.com Text Role of the renin-angiotensin system in NETosis in the coronavirus disease 2019 (COVID-19) JO: Biomed Pharmacother http://www.kidney.de/pget.php?&tw=1&pmid=35176710 #FOAvip Myocardial infarction and stroke are the leading causes of death in the world. Numerous evidence has confirmed that hypertension promotes thrombosis and induces myocardial infarction and stroke. Recent findings reveal that neutrophil extracellular traps (NETs) are involved in the induction of myocardial infarction and stroke. Meanwhile, patients with severe COVID-19 suffer from complications such as myocardial infarction and stroke with pathological signs of NETs. Due to the extremely low amount of virus detected in the blood and remote organs (e.g., heart, brain and kidney) in a few cases, it is difficult to explain the mechanism by which the virus triggers NETosis, and there may be a different mechanism than in the lung. A large number of studies have found that the renin-angiotensin system regulates the NETosis at multiple levels in patients with COVID-19, such as endocytosis of SARS-COV-2, abnormal angiotensin II levels, neutrophil activation and procoagulant function at multiple levels, which may contribute to the formation of reticular structure and thrombosis. The treatment of angiotensin-converting enzyme inhibitors (ACEI), angiotensin II type 1 receptor blockers (ARBs) and neutrophil recruitment and active antagonists helps to regulate blood pressure and reduce the risk of net and thrombosis. The review will explore the possible role of the angiotensin system in the formation of NETs in severe COVID-19. Twit Text FOAVip Role of the renin-angiotensin system in NETosis in the coronavirus disease 2019 (COVID-19) ????Biomed Pharmacother http://www.kidney.de/pget.php?&tw=1&pmid=35176710 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=35176710 #FOAvip English Wikipedia <ref>{{Cite pmid|35176710}}</ref> Role of the renin-angiotensin system in NETosis in the coronavirus disease 2019 (COVID-19) #MMPMID35176710 Zhang Q; Ling S; Hu K; Liu J; Xu JW Biomed Pharmacother 2022[Apr]; 148 (ä): 112718 PMID35176710show ga Myocardial infarction and stroke are the leading causes of death in the world. Numerous evidence has confirmed that hypertension promotes thrombosis and induces myocardial infarction and stroke. Recent findings reveal that neutrophil extracellular traps (NETs) are involved in the induction of myocardial infarction and stroke. Meanwhile, patients with severe COVID-19 suffer from complications such as myocardial infarction and stroke with pathological signs of NETs. Due to the extremely low amount of virus detected in the blood and remote organs (e.g., heart, brain and kidney) in a few cases, it is difficult to explain the mechanism by which the virus triggers NETosis, and there may be a different mechanism than in the lung. A large number of studies have found that the renin-angiotensin system regulates the NETosis at multiple levels in patients with COVID-19, such as endocytosis of SARS-COV-2, abnormal angiotensin II levels, neutrophil activation and procoagulant function at multiple levels, which may contribute to the formation of reticular structure and thrombosis. The treatment of angiotensin-converting enzyme inhibitors (ACEI), angiotensin II type 1 receptor blockers (ARBs) and neutrophil recruitment and active antagonists helps to regulate blood pressure and reduce the risk of net and thrombosis. The review will explore the possible role of the angiotensin system in the formation of NETs in severe COVID-19. |*COVID-19[MESH] |*Extracellular Traps[MESH] |*Neutrophils[MESH] |*Renin-Angiotensin System[MESH] |*SARS-CoV-2[MESH] |Angiotensin II[MESH] |Animals[MESH] |Humans[MESH] |Peptidyl-Dipeptidase A[MESH]Role of the renin-angiotensin system in NETosis in the coronavirus dis(epmc).pdf DeepDyve Pubget Overpricing