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10.3389/fimmu.2022.732197 http://scihub22266oqcxt.onion/10.3389/fimmu.2022.732197 35154090!8831226!35154090     free     free     free Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Front+Immunol 2022 ; 13 (ä): 732197 Nephropedia Template TP {{tp|p=35154090|t=2022. SARS-CoV-2 Infection Triggers Auto-Immune Response in ARDS |pdf=|usr=}}{{35154090}} gab.com Text SARS-CoV-2 Infection Triggers Auto-Immune Response in ARDS JO: Front Immunol http://www.kidney.de/pget.php?&tw=1&pmid=35154090 #FOAvip Acute respiratory distress syndrome (ARDS) is a severe pulmonary disease, which is one of the major complications in COVID-19 patients. Dysregulation of the immune system and imbalances in cytokine release and immune cell activation are involved in SARS-CoV-2 infection. Here, the inflammatory, antigen, and auto-immune profile of patients presenting COVID-19-associated severe ARDS has been analyzed using functional proteomics approaches. Both, innate and humoral responses have been characterized through acute-phase protein network and auto-antibody signature. Severity and sepsis by SARS-CoV-2 emerged to be correlated with auto-immune profiles of patients and define their clinical progression, which could provide novel perspectives in therapeutics development and biomarkers of COVID-19 patients. Humoral response in COVID-19 patients' profile separates with significant differences patients with or without ARDS. Furthermore, we found that this profile can be correlated with COVID-19 severity and results more common in elderly patients. Twit Text FOAVip SARS-CoV-2 Infection Triggers Auto-Immune Response in ARDS ????Front Immunol http://www.kidney.de/pget.php?&tw=1&pmid=35154090 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=35154090 #FOAvip English Wikipedia <ref>{{Cite pmid|35154090}}</ref> SARS-CoV-2 Infection Triggers Auto-Immune Response in ARDS #MMPMID35154090 Juanes-Velasco P; Landeira-Vinuela A; Garcia-Vaquero ML; Lecrevisse Q; Herrero R; Ferruelo A; Gongora R; Corrales F; Rivas JL; Lorente JA; Hernandez AP; Fuentes M Front Immunol 2022[]; 13 (ä): 732197 PMID35154090show ga Acute respiratory distress syndrome (ARDS) is a severe pulmonary disease, which is one of the major complications in COVID-19 patients. Dysregulation of the immune system and imbalances in cytokine release and immune cell activation are involved in SARS-CoV-2 infection. Here, the inflammatory, antigen, and auto-immune profile of patients presenting COVID-19-associated severe ARDS has been analyzed using functional proteomics approaches. Both, innate and humoral responses have been characterized through acute-phase protein network and auto-antibody signature. Severity and sepsis by SARS-CoV-2 emerged to be correlated with auto-immune profiles of patients and define their clinical progression, which could provide novel perspectives in therapeutics development and biomarkers of COVID-19 patients. Humoral response in COVID-19 patients' profile separates with significant differences patients with or without ARDS. Furthermore, we found that this profile can be correlated with COVID-19 severity and results more common in elderly patients. |Autoantibodies/immunology[MESH] |Autoantigens/*immunology[MESH] |Autoimmunity/*immunology[MESH] |COVID-19/complications/*immunology[MESH] |Humans[MESH] |Respiratory Distress Syndrome/*immunology/*virology[MESH]SARS-CoV-2 Infection Triggers Auto-Immune Response in ARDS (epmc).pdf DeepDyve Pubget Overpricing