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10.1007/s12275-022-1525-1

http://scihub22266oqcxt.onion/10.1007/s12275-022-1525-1
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35122601!8817151!35122601
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suck abstract from ncbi


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pmid35122601      J+Microbiol 2022 ; 60 (3): 290-299
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  • SARS-CoV-2-mediated evasion strategies for antiviral interferon pathways #MMPMID35122601
  • Oh SJ; Shin OS
  • J Microbiol 2022[Mar]; 60 (3): 290-299 PMID35122601show ga
  • With global expansion of the COVID-19 pandemic and the emergence of new variants, extensive efforts have been made to develop highly effective antiviral drugs and vaccines against SARS-CoV-2. The interactions of coronaviruses with host antiviral interferon pathways ultimately determine successful viral replication and SARS-CoV-2-induced pathogenesis. Innate immune receptors play an essential role in host defense against SARS-CoV-2 via the induction of IFN production and signaling. Here, we summarize the recent advances in innate immune sensing mechanisms of SARS-CoV-2 and various strategies by which SARS-CoV-2 antagonizes antiviral innate immune signaling pathways, with a particular focus on mechanisms utilized by multiple SARS-CoV-2 proteins to evade interferon induction and signaling in host cell. Understanding the underlying immune evasion mechanisms of SARS-CoV-2 is essential for the improvement of vaccines and therapeutic strategies.
  • |*COVID-19/immunology/virology[MESH]
  • |*Immune Evasion[MESH]
  • |Antiviral Restriction Factors/immunology[MESH]
  • |Humans[MESH]
  • |Immunity, Innate[MESH]
  • |Interferons/*immunology[MESH]
  • |Pandemics[MESH]


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