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10.1007/s00281-022-00916-w http://scihub22266oqcxt.onion/10.1007/s00281-022-00916-w 35122116!8816310!35122116     free     free     free Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Semin+Immunopathol 2022 ; 44 (3): 347-362 Nephropedia Template TP {{tp|p=35122116|t=2022. Mechanisms of immunothrombosis and vasculopathy in antiphospholipid syndrome |pdf=|usr=}}{{35122116}} gab.com Text Mechanisms of immunothrombosis and vasculopathy in antiphospholipid syndrome JO: Semin Immunopathol http://www.kidney.de/pget.php?&tw=1&pmid=35122116 #FOAvip Antiphospholipid syndrome (APS) is an autoimmune thrombophilia propelled by circulating antiphospholipid antibodies that herald vascular thrombosis and obstetrical complications. Antiphospholipid antibodies recognize phospholipids and phospholipid-binding proteins and are not only markers of disease but also key drivers of APS pathophysiology. Thrombotic events in APS can be attributed to various conspirators including activated endothelial cells, platelets, and myeloid-lineage cells, as well as derangements in coagulation and fibrinolytic systems. Furthermore, recent work has especially highlighted the role of neutrophil extracellular traps (NETs) and the complement system in APS thrombosis. Beyond acute thrombosis, patients with APS can also develop an occlusive vasculopathy, a long-term consequence of APS characterized by cell proliferation and infiltration that progressively expands the intima and leads to organ damage. This review will highlight known pathogenic factors in APS and will also briefly discuss similarities between APS and the thrombophilic coagulopathy of COVID-19. Twit Text FOAVip Mechanisms of immunothrombosis and vasculopathy in antiphospholipid syndrome ????Semin Immunopathol http://www.kidney.de/pget.php?&tw=1&pmid=35122116 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=35122116 #FOAvip English Wikipedia <ref>{{Cite pmid|35122116}}</ref> Mechanisms of immunothrombosis and vasculopathy in antiphospholipid syndrome #MMPMID35122116 Knight JS; Kanthi Y Semin Immunopathol 2022[May]; 44 (3): 347-362 PMID35122116show ga Antiphospholipid syndrome (APS) is an autoimmune thrombophilia propelled by circulating antiphospholipid antibodies that herald vascular thrombosis and obstetrical complications. Antiphospholipid antibodies recognize phospholipids and phospholipid-binding proteins and are not only markers of disease but also key drivers of APS pathophysiology. Thrombotic events in APS can be attributed to various conspirators including activated endothelial cells, platelets, and myeloid-lineage cells, as well as derangements in coagulation and fibrinolytic systems. Furthermore, recent work has especially highlighted the role of neutrophil extracellular traps (NETs) and the complement system in APS thrombosis. Beyond acute thrombosis, patients with APS can also develop an occlusive vasculopathy, a long-term consequence of APS characterized by cell proliferation and infiltration that progressively expands the intima and leads to organ damage. This review will highlight known pathogenic factors in APS and will also briefly discuss similarities between APS and the thrombophilic coagulopathy of COVID-19. |*Antiphospholipid Syndrome/complications/diagnosis[MESH] |*COVID-19/complications[MESH] |*Thrombosis/complications[MESH] |*Vascular Diseases[MESH] |Antibodies, Antiphospholipid[MESH] |Endothelial Cells[MESH] |Humans[MESH]Mechanisms of immunothrombosis and vasculopathy in antiphospholipid sy(epmc).pdf DeepDyve Pubget Overpricing