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10.1007/s12275-022-1502-8

http://scihub22266oqcxt.onion/10.1007/s12275-022-1502-8
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35089584!8795727!35089584
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suck abstract from ncbi


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pmid35089584      J+Microbiol 2022 ; 60 (3): 300-307
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  • Coordinated regulation of interferon and inflammasome signaling pathways by SARS-CoV-2 proteins #MMPMID35089584
  • Kim NE; Song YJ
  • J Microbiol 2022[Mar]; 60 (3): 300-307 PMID35089584show ga
  • Type I and III interferons (IFNs) and the nucleotide-binding domain (NBD) leucine-rich repeat (LRR)-containing receptor (NLR) family pyrin domain containing 3 (NLRP3) inflammasome play pivotal roles in the pathogenesis of SARS-CoV-2. While optimal IFN and inflammasome responses are essential for limiting SARS-CoV-2 infection, aberrant activation of these innate immune responses is associated with COVID-19 pathogenesis. In this review, we focus our discussion on recent findings on SARS-CoV-2-induced type I and III IFNs and NLRP3 inflammasome responses and the viral proteins regulating these mechanisms.
  • |*COVID-19/immunology[MESH]
  • |*Inflammasomes/metabolism[MESH]
  • |*NLR Family, Pyrin Domain-Containing 3 Protein/metabolism[MESH]
  • |Humans[MESH]
  • |Interferons/*immunology[MESH]
  • |SARS-CoV-2[MESH]


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