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10.1016/j.cytogfr.2022.01.002

http://scihub22266oqcxt.onion/10.1016/j.cytogfr.2022.01.002
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35039221!8741331!35039221
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suck abstract from ncbi


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pmid35039221      Cytokine+Growth+Factor+Rev 2022 ; 63 (ä): 108-118
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  • Before the "cytokine storm": Boosting efferocytosis as an effective strategy against SARS-CoV-2 infection and associated complications #MMPMID35039221
  • Dutta S; Mukherjee A; Nongthomba U
  • Cytokine Growth Factor Rev 2022[Feb]; 63 (ä): 108-118 PMID35039221show ga
  • The Severe Acute Respiratory Syndrome Coronavirus 2 (SARS-CoV-2) is responsible for the ongoing COVID-19 pandemic, and causes many health complications, including major lung diseases. Besides investigations into the virology of SARS-CoV-2, understanding the immunological routes underlying the clinical manifestations of COVID-19 is important for developing effective therapeutic interventions. The clearance of SARS-CoV-2-infected apoptotic cells by professional efferocytes, through a process termed as 'efferocytosis', is essential for maintaining tissue homeostasis, and reducing the chances of health complications caused by SARS-CoV-2 infection. In this review, we focus on the cellular events leading to engagement of the SARS-CoV-2 with type 2 alveolar cells, and how SARS-COV-2 infection impairs the macrophage anti-inflammatory programming. We also discuss accounts of impaired efferocytosis, and the "cytokine storm" which occur concomitantly with the SARS-CoV-2 infection. Finally, we propose how targeting impaired efferocytosis, due to the SARS-CoV-2 infection, may be a beneficial therapeutic strategy to combat COVID-19, and its complications.
  • |*COVID-19[MESH]
  • |Cytokine Release Syndrome[MESH]
  • |Humans[MESH]
  • |Macrophages[MESH]
  • |Pandemics[MESH]


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