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10.1371/journal.ppat.1010161

http://scihub22266oqcxt.onion/10.1371/journal.ppat.1010161
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35025969!8757994!35025969
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suck abstract from ncbi

pmid35025969      PLoS+Pathog 2022 ; 18 (1): e1010161
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  • Advances and gaps in SARS-CoV-2 infection models #MMPMID35025969
  • Munoz-Fontela C; Widerspick L; Albrecht RA; Beer M; Carroll MW; de Wit E; Diamond MS; Dowling WE; Funnell SGP; Garcia-Sastre A; Gerhards NM; de Jong R; Munster VJ; Neyts J; Perlman S; Reed DS; Richt JA; Riveros-Balta X; Roy CJ; Salguero FJ; Schotsaert M; Schwartz LM; Seder RA; Segales J; Vasan SS; Henao-Restrepo AM; Barouch DH
  • PLoS Pathog 2022[Jan]; 18 (1): e1010161 PMID35025969show ga
  • The global response to Coronavirus Disease 2019 (COVID-19) is now facing new challenges such as vaccine inequity and the emergence of SARS-CoV-2 variants of concern (VOCs). Preclinical models of disease, in particular animal models, are essential to investigate VOC pathogenesis, vaccine correlates of protection and postexposure therapies. Here, we provide an update from the World Health Organization (WHO) COVID-19 modeling expert group (WHO-COM) assembled by WHO, regarding advances in preclinical models. In particular, we discuss how animal model research is playing a key role to evaluate VOC virulence, transmission and immune escape, and how animal models are being refined to recapitulate COVID-19 demographic variables such as comorbidities and age.
  • |*Disease Models, Animal[MESH]
  • |*SARS-CoV-2/immunology/pathogenicity[MESH]
  • |Age Factors[MESH]
  • |Animals[MESH]
  • |COVID-19 Vaccines/adverse effects/immunology[MESH]
  • |COVID-19/*etiology/prevention & control/therapy[MESH]
  • |Comorbidity[MESH]


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