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10.1016/j.molmed.2021.11.004 http://scihub22266oqcxt.onion/10.1016/j.molmed.2021.11.004 34922835!8673590!34922835     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Trends+Mol+Med 2022 ; 28 (2): 143-154 Nephropedia Template TP {{tp|p=34922835|t=2022. Understanding the tuberculosis granuloma: the matrix revolutions |pdf=|usr=}}{{34922835}} gab.com Text Understanding the tuberculosis granuloma: the matrix revolutions JO: Trends Mol Med http://www.kidney.de/pget.php?&tw=1&pmid=34922835 #FOAvip Mycobacterium tuberculosis (Mtb) causes the human disease tuberculosis (TB) and remains the top global infectious pandemic after coronavirus disease 2019 (COVID-19). Furthermore, TB has killed many more humans than any other pathogen, after prolonged coevolution to optimise its pathogenic strategies. Full understanding of fundamental disease processes in humans is necessary to successfully combat this highly successful pathogen. While the importance of immunodeficiency has been long recognised, biologic therapies and unbiased approaches are providing unprecedented insights into the intricacy of the host-pathogen interaction. The nature of a protective response is more complex than previously hypothesised. Here, we integrate recent evidence from human studies and unbiased approaches to consider how Mtb causes human TB and highlight the recurring theme of extracellular matrix (ECM) turnover. Twit Text FOAVip Understanding the tuberculosis granuloma: the matrix revolutions ????Trends Mol Med http://www.kidney.de/pget.php?&tw=1&pmid=34922835 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=34922835 #FOAvip English Wikipedia <ref>{{Cite pmid|34922835}}</ref> Understanding the tuberculosis granuloma: the matrix revolutions #MMPMID34922835 Elkington P; Polak ME; Reichmann MT; Leslie A Trends Mol Med 2022[Feb]; 28 (2): 143-154 PMID34922835show ga Mycobacterium tuberculosis (Mtb) causes the human disease tuberculosis (TB) and remains the top global infectious pandemic after coronavirus disease 2019 (COVID-19). Furthermore, TB has killed many more humans than any other pathogen, after prolonged coevolution to optimise its pathogenic strategies. Full understanding of fundamental disease processes in humans is necessary to successfully combat this highly successful pathogen. While the importance of immunodeficiency has been long recognised, biologic therapies and unbiased approaches are providing unprecedented insights into the intricacy of the host-pathogen interaction. The nature of a protective response is more complex than previously hypothesised. Here, we integrate recent evidence from human studies and unbiased approaches to consider how Mtb causes human TB and highlight the recurring theme of extracellular matrix (ECM) turnover. |*COVID-19[MESH] |*Mycobacterium tuberculosis[MESH] |*Tuberculosis[MESH] |Granuloma[MESH] |Host-Pathogen Interactions[MESH] |Humans[MESH]Understanding the tuberculosis granuloma: the matrix revolutions (epmc).pdf DeepDyve Pubget Overpricing