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10.1016/j.mayocp.2021.06.027 http://scihub22266oqcxt.onion/10.1016/j.mayocp.2021.06.027 34863398!8373818!34863398     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Mayo+Clin+Proc 2021 ; 96 (12): 3099-3108 Nephropedia Template TP {{tp|p=34863398|t=2021. Viral Endothelial Dysfunction: A Unifying Mechanism for COVID-19 |pdf=|usr=}}{{34863398}} gab.com Text Viral Endothelial Dysfunction: A Unifying Mechanism for COVID-19 JO: Mayo Clin Proc http://www.kidney.de/pget.php?&tw=1&pmid=34863398 #FOAvip The severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) is a highly transmissible virus with significant global impact, morbidity, and mortality. The SARS-CoV-2 virus may result in widespread organ manifestations including acute respiratory distress syndrome, acute renal failure, thromboembolism, and myocarditis. Virus-induced endothelial injury may cause endothelial activation, increased permeability, inflammation, and immune response and cytokine storm. Endothelial dysfunction is a systemic disorder that is a precursor of atherosclerotic vascular disease that is associated with cardiovascular risk factors and is highly prevalent in patients with atherosclerotic cardiovascular and peripheral disease. Several studies have associated various viral infections including SARS-CoV-2 infection with inflammation, endothelial dysfunction, and subsequent innate immune response and cytokine storm. Noninvasive monitoring of endothelial function and identification of high-risk patients who may require specific therapies may have the potential to improve morbidity and mortality associated with subsequent inflammation, cytokine storm, and multiorgan involvement. Twit Text FOAVip Viral Endothelial Dysfunction: A Unifying Mechanism for COVID-19 ????Mayo Clin Proc http://www.kidney.de/pget.php?&tw=1&pmid=34863398 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=34863398 #FOAvip English Wikipedia <ref>{{Cite pmid|34863398}}</ref> Viral Endothelial Dysfunction: A Unifying Mechanism for COVID-19 #MMPMID34863398 Prasad M; Leon M; Lerman LO; Lerman A Mayo Clin Proc 2021[Dec]; 96 (12): 3099-3108 PMID34863398show ga The severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) is a highly transmissible virus with significant global impact, morbidity, and mortality. The SARS-CoV-2 virus may result in widespread organ manifestations including acute respiratory distress syndrome, acute renal failure, thromboembolism, and myocarditis. Virus-induced endothelial injury may cause endothelial activation, increased permeability, inflammation, and immune response and cytokine storm. Endothelial dysfunction is a systemic disorder that is a precursor of atherosclerotic vascular disease that is associated with cardiovascular risk factors and is highly prevalent in patients with atherosclerotic cardiovascular and peripheral disease. Several studies have associated various viral infections including SARS-CoV-2 infection with inflammation, endothelial dysfunction, and subsequent innate immune response and cytokine storm. Noninvasive monitoring of endothelial function and identification of high-risk patients who may require specific therapies may have the potential to improve morbidity and mortality associated with subsequent inflammation, cytokine storm, and multiorgan involvement. |*COVID-19/immunology/physiopathology[MESH] |*Endothelium/physiopathology/virology[MESH] |Cytokine Release Syndrome/virology[MESH] |Disease Management[MESH] |Humans[MESH] |Multiple Organ Failure/etiology/immunology[MESH] |SARS-CoV-2/physiology[MESH]Viral Endothelial Dysfunction: A Unifying Mechanism for COVID-19 (epmc).pdf DeepDyve Pubget Overpricing