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10.3389/fimmu.2021.752380 http://scihub22266oqcxt.onion/10.3389/fimmu.2021.752380 34691068!8531724!34691068     free     free     free Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Front+Immunol 2021 ; 12 (ä): 752380 Nephropedia Template TP {{tp|p=34691068|t=2021. COVID-19 Is a Multi-Organ Aggressor: Epigenetic and Clinical Marks |pdf=|usr=}}{{34691068}} gab.com Text COVID-19 Is a Multi-Organ Aggressor: Epigenetic and Clinical Marks JO: Front Immunol http://www.kidney.de/pget.php?&tw=1&pmid=34691068 #FOAvip The progression of coronavirus disease 2019 (COVID-19), resulting from a severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection, may be influenced by both genetic and environmental factors. Several viruses hijack the host genome machinery for their own advantage and survival, and similar phenomena might occur upon SARS-CoV-2 infection. Severe cases of COVID-19 may be driven by metabolic and epigenetic driven mechanisms, including DNA methylation and histone/chromatin alterations. These epigenetic phenomena may respond to enhanced viral replication and mediate persistent long-term infection and clinical phenotypes associated with severe COVID-19 cases and fatalities. Understanding the epigenetic events involved, and their clinical significance, may provide novel insights valuable for the therapeutic control and management of the COVID-19 pandemic. This review highlights different epigenetic marks potentially associated with COVID-19 development, clinical manifestation, and progression. Twit Text FOAVip COVID-19 Is a Multi-Organ Aggressor: Epigenetic and Clinical Marks ????Front Immunol http://www.kidney.de/pget.php?&tw=1&pmid=34691068 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=34691068 #FOAvip English Wikipedia <ref>{{Cite pmid|34691068}}</ref> COVID-19 Is a Multi-Organ Aggressor: Epigenetic and Clinical Marks #MMPMID34691068 Kgatle MM; Lawal IO; Mashabela G; Boshomane TMG; Koatale PC; Mahasha PW; Ndlovu H; Vorster M; Rodrigues HG; Zeevaart JR; Gordon S; Moura-Alves P; Sathekge MM Front Immunol 2021[]; 12 (ä): 752380 PMID34691068show ga The progression of coronavirus disease 2019 (COVID-19), resulting from a severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection, may be influenced by both genetic and environmental factors. Several viruses hijack the host genome machinery for their own advantage and survival, and similar phenomena might occur upon SARS-CoV-2 infection. Severe cases of COVID-19 may be driven by metabolic and epigenetic driven mechanisms, including DNA methylation and histone/chromatin alterations. These epigenetic phenomena may respond to enhanced viral replication and mediate persistent long-term infection and clinical phenotypes associated with severe COVID-19 cases and fatalities. Understanding the epigenetic events involved, and their clinical significance, may provide novel insights valuable for the therapeutic control and management of the COVID-19 pandemic. This review highlights different epigenetic marks potentially associated with COVID-19 development, clinical manifestation, and progression. |COVID-19/genetics/*immunology[MESH] |DNA Methylation/*immunology[MESH] |Epigenesis, Genetic/*immunology[MESH] |Humans[MESH] |Organ Specificity[MESH] |Pandemics[MESH]COVID-19 Is a Multi-Organ Aggressor: Epigenetic and Clinical Marks (epmc).pdf DeepDyve Pubget Overpricing