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10.3390/ijms222011279

http://scihub22266oqcxt.onion/10.3390/ijms222011279
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34681944!8538208!34681944
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suck abstract from ncbi


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pmid34681944      Int+J+Mol+Sci 2021 ; 22 (20): ä
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  • Alveolar Regeneration in COVID-19 Patients: A Network Perspective #MMPMID34681944
  • Gupta SK; Srivastava M; Minocha R; Akash A; Dangwal S; Dandekar T
  • Int J Mol Sci 2021[Oct]; 22 (20): ä PMID34681944show ga
  • A viral infection involves entry and replication of viral nucleic acid in a host organism, subsequently leading to biochemical and structural alterations in the host cell. In the case of SARS-CoV-2 viral infection, over-activation of the host immune system may lead to lung damage. Albeit the regeneration and fibrotic repair processes being the two protective host responses, prolonged injury may lead to excessive fibrosis, a pathological state that can result in lung collapse. In this review, we discuss regeneration and fibrosis processes in response to SARS-CoV-2 and provide our viewpoint on the triggering of alveolar regeneration in coronavirus disease 2019 (COVID-19) patients.
  • |*Regeneration[MESH]
  • |COVID-19/*pathology/virology[MESH]
  • |Epigenomics[MESH]
  • |Fibrosis[MESH]
  • |Humans[MESH]
  • |Immune System/metabolism[MESH]
  • |Lung/*physiology[MESH]
  • |MicroRNAs/metabolism[MESH]
  • |SARS-CoV-2/isolation & purification[MESH]


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