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10.1016/j.clim.2021.108842 http://scihub22266oqcxt.onion/10.1016/j.clim.2021.108842 34461289!8393504!34461289     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=34461289&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Clin+Immunol 2021 ; 231 (?): 108842 Nephropedia Template TP {{tp|p=34461289|t=2021. Immunomodulation and immunotherapeutics of COVID-19 |pdf=|usr=}}{{34461289}} gab.com Text Immunomodulation and immunotherapeutics of COVID-19 JO: Clin Immunol http://www.kidney.de/pget.php?&tw=1&pmid=34461289 #FOAvip Severe acute respiratory syndrome coronavirus-2 causes coronavirus disease 2019, a pandemic which was originated from Wuhan city of China. The pandemic has affected millions of people worldwide. The pathogenesis of SARS-CoV-2 is characterized by a cytokine storm in the blood (cytokinemia) and tissues, especially the lungs. One of the major repercussions of this inflammatory process is the endothelial injury-causing intestinal bleeding, coagulopathy, and thromboembolism which result in various sudden and unexpected post-COVID complications including kidney failure, myocardial infarction, or multiorgan failure. In this review, we have summarized the immune responses, biochemical changes, and inflammatory responses in the human body after infection with the SARS-CoV-2 virus. The increased amount of inflammatory cytokines, chemokines, and involvement of complement proteins in inflammatory reaction increase the risk of occurrence of disease. Twit Text FOAVip Immunomodulation and immunotherapeutics of COVID-19 ????Clin Immunol http://www.kidney.de/pget.php?&tw=1&pmid=34461289 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=34461289 #FOAvip English Wikipedia <ref>{{Cite pmid|34461289}}</ref> Immunomodulation and immunotherapeutics of COVID-19 #MMPMID34461289 Garg SS; Sharma A; Gupta J Clin Immunol 2021[Oct]; 231 (?): 108842 PMID34461289show ga Severe acute respiratory syndrome coronavirus-2 causes coronavirus disease 2019, a pandemic which was originated from Wuhan city of China. The pandemic has affected millions of people worldwide. The pathogenesis of SARS-CoV-2 is characterized by a cytokine storm in the blood (cytokinemia) and tissues, especially the lungs. One of the major repercussions of this inflammatory process is the endothelial injury-causing intestinal bleeding, coagulopathy, and thromboembolism which result in various sudden and unexpected post-COVID complications including kidney failure, myocardial infarction, or multiorgan failure. In this review, we have summarized the immune responses, biochemical changes, and inflammatory responses in the human body after infection with the SARS-CoV-2 virus. The increased amount of inflammatory cytokines, chemokines, and involvement of complement proteins in inflammatory reaction increase the risk of occurrence of disease. |*Immunomodulation[MESH] |*Immunotherapy[MESH] |*SARS-CoV-2[MESH] |COVID-19/*immunology/pathology/*therapy[MESH] |Humans[MESH]Immunomodulation and immunotherapeutics of COVID-19 (epmc).pdf DeepDyve Pubget Overpricing