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10.3390/cells10081993 http://scihub22266oqcxt.onion/10.3390/cells10081993 34440767!8391332!34440767     free     free     free Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Cells 2021 ; 10 (8): ä Nephropedia Template TP {{tp|p=34440767|t=2021. Microbiota-Gut-Brain Communication in the SARS-CoV-2 Infection |pdf=|usr=}}{{34440767}} gab.com Text Microbiota-Gut-Brain Communication in the SARS-CoV-2 Infection JO: Cells http://www.kidney.de/pget.php?&tw=1&pmid=34440767 #FOAvip The coronavirus disease of 2019 (COVID-19) is an infectious disease caused by severe acute respiratory syndrome 2 (SARS-CoV-2). In addition to pneumonia, individuals affected by the disease have neurological symptoms. Indeed, SARS-CoV-2 has a neuroinvasive capacity. It is known that the infection caused by SARS-CoV-2 leads to a cytokine storm. An exacerbated inflammatory state can lead to the blood-brain barrier (BBB) damage as well as to intestinal dysbiosis. These changes, in turn, are associated with microglial activation and reactivity of astrocytes that can promote the degeneration of neurons and be associated with the development of psychiatric disorders and neurodegenerative diseases. Studies also have been shown that SARS-CoV-2 alters the composition and functional activity of the gut microbiota. The microbiota-gut-brain axis provides a bidirectional homeostatic communication pathway. Thus, this review focuses on studies that show the relationship between inflammation and the gut microbiota-brain axis in SARS-CoV-2 infection. Twit Text FOAVip Microbiota-Gut-Brain Communication in the SARS-CoV-2 Infection ????Cells http://www.kidney.de/pget.php?&tw=1&pmid=34440767 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=34440767 #FOAvip English Wikipedia <ref>{{Cite pmid|34440767}}</ref> Microbiota-Gut-Brain Communication in the SARS-CoV-2 Infection #MMPMID34440767 Manosso LM; Arent CO; Borba LA; Ceretta LB; Quevedo J; Reus GZ Cells 2021[Aug]; 10 (8): ä PMID34440767show ga The coronavirus disease of 2019 (COVID-19) is an infectious disease caused by severe acute respiratory syndrome 2 (SARS-CoV-2). In addition to pneumonia, individuals affected by the disease have neurological symptoms. Indeed, SARS-CoV-2 has a neuroinvasive capacity. It is known that the infection caused by SARS-CoV-2 leads to a cytokine storm. An exacerbated inflammatory state can lead to the blood-brain barrier (BBB) damage as well as to intestinal dysbiosis. These changes, in turn, are associated with microglial activation and reactivity of astrocytes that can promote the degeneration of neurons and be associated with the development of psychiatric disorders and neurodegenerative diseases. Studies also have been shown that SARS-CoV-2 alters the composition and functional activity of the gut microbiota. The microbiota-gut-brain axis provides a bidirectional homeostatic communication pathway. Thus, this review focuses on studies that show the relationship between inflammation and the gut microbiota-brain axis in SARS-CoV-2 infection. |Brain/*physiology[MESH] |COVID-19/*physiopathology[MESH] |Dysbiosis[MESH] |Gastrointestinal Microbiome/*physiology[MESH] |Humans[MESH] |Inflammation[MESH] |Mood Disorders[MESH]Microbiota-Gut-Brain Communication in the SARS-CoV-2 Infection (epmc).pdf DeepDyve Pubget Overpricing