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10.1097/INF.0000000000003295 http://scihub22266oqcxt.onion/10.1097/INF.0000000000003295 34387618!8505151!34387618     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=34387618&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Pediatr+Infect+Dis+J 2021 ; 40 (11): e445-e450 Nephropedia Template TP {{tp|p=34387618|t=2021. COVID-19-associated Acute Disseminated Encephalomyelitis-like Disease in 2 Children |pdf=|usr=}}{{34387618}} gab.com Text COVID-19-associated Acute Disseminated Encephalomyelitis-like Disease in 2 Children JO: Pediatr Infect Dis J http://www.kidney.de/pget.php?&tw=1&pmid=34387618 #FOAvip BACKGROUND: The coronavirus disease 2019 pandemic was caused by the novel severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2). Although the predominant clinical presentation is a respiratory disease, neurologic manifestations are being recognized increasingly. CASE REPORT: We report 2 children 9 years of age who developed acute disseminated encephalomyelitis-like disease associated with SARS-CoV-2. Seizures and encephalopathy were the main central nervous system symptoms. The cerebrospinal fluid analysis performed within the first week of disease onset showed elevated protein in both children with normal cell count and no evidence of infection including negative SARS-CoV-2 by antibody and polymerase chain reaction. Brain magnetic resonance imaging revealed T2A, fluid-attenuated inversion recovery cortical and subcortical hyperintensity without restricted diffusion consistent with acute disseminated encephalomyelitis-like disease. They received methylprednisolone followed by therapeutic plasma exchange. One of them showed complete clinical improvement and resolution in magnetic resonance imaging findings. The other developed laminar necrosis in brain magnetic resonance imaging and showed significant clinical improvement after therapeutic plasma exchange. He was positive for positive SARS-CoV-2 antibody in cerebrospinal fluid on day 55 of admission. They were both positive for SARS-CoV-2 antibodies in serum after 2 weeks. CONCLUSIONS: Our two cases highlight the occurrence of acute disseminated encephalomyelitis-like disease as a postinfectious/immune-mediated complication of SARS-CoV-2 infection. Twit Text FOAVip COVID-19-associated Acute Disseminated Encephalomyelitis-like Disease in 2 Children ????Pediatr Infect Dis J http://www.kidney.de/pget.php?&tw=1&pmid=34387618 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=34387618 #FOAvip English Wikipedia <ref>{{Cite pmid|34387618}}</ref> COVID-19-associated Acute Disseminated Encephalomyelitis-like Disease in 2 Children #MMPMID34387618 Akcay N; Bektas G; Menentoglu ME; Ogur M; Sofuoglu AI; Palabiyik FB; Sevketoglu E Pediatr Infect Dis J 2021[Nov]; 40 (11): e445-e450 PMID34387618show ga BACKGROUND: The coronavirus disease 2019 pandemic was caused by the novel severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2). Although the predominant clinical presentation is a respiratory disease, neurologic manifestations are being recognized increasingly. CASE REPORT: We report 2 children 9 years of age who developed acute disseminated encephalomyelitis-like disease associated with SARS-CoV-2. Seizures and encephalopathy were the main central nervous system symptoms. The cerebrospinal fluid analysis performed within the first week of disease onset showed elevated protein in both children with normal cell count and no evidence of infection including negative SARS-CoV-2 by antibody and polymerase chain reaction. Brain magnetic resonance imaging revealed T2A, fluid-attenuated inversion recovery cortical and subcortical hyperintensity without restricted diffusion consistent with acute disseminated encephalomyelitis-like disease. They received methylprednisolone followed by therapeutic plasma exchange. One of them showed complete clinical improvement and resolution in magnetic resonance imaging findings. The other developed laminar necrosis in brain magnetic resonance imaging and showed significant clinical improvement after therapeutic plasma exchange. He was positive for positive SARS-CoV-2 antibody in cerebrospinal fluid on day 55 of admission. They were both positive for SARS-CoV-2 antibodies in serum after 2 weeks. CONCLUSIONS: Our two cases highlight the occurrence of acute disseminated encephalomyelitis-like disease as a postinfectious/immune-mediated complication of SARS-CoV-2 infection. |*SARS-CoV-2[MESH] |Biomarkers[MESH] |COVID-19/*complications/*virology[MESH] |Disease Management[MESH] |Disease Susceptibility[MESH] |Electroencephalography[MESH] |Encephalomyelitis, Acute Disseminated/blood/*diagnosis/*etiology/therapy[MESH] |Female[MESH] |Humans[MESH] |Immunoglobulins, Intravenous/therapeutic use[MESH] |Magnetic Resonance Imaging[MESH] |Male[MESH] |Symptom Assessment[MESH] |Treatment Outcome[MESH]COVID-19-associated Acute Disseminated Encephalomyelitis-like Disease (epmc).pdf DeepDyve Pubget Overpricing