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10.1080/09537104.2021.1962519

http://scihub22266oqcxt.onion/10.1080/09537104.2021.1962519
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34384321!8425443!34384321
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suck abstract from ncbi

pmid34384321      Platelets 2022 ; 33 (2): 192-199
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  • Alterations in platelets during SARS-CoV-2 infection #MMPMID34384321
  • Brambilla M; Canzano P; Becchetti A; Tremoli E; Camera M
  • Platelets 2022[Feb]; 33 (2): 192-199 PMID34384321show ga
  • Coronavirus disease 2019 (COVID-19) is a pandemic syndrome caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection. SARS-CoV-2 infection induces a process of inflammation and thrombosis supported by an altered platelet activation state. This platelet activation is peculiar being characterized by the formation of platelet-leukocytes rather than platelet-platelet aggregates and by an increased procoagulant potential supported by elevated levels of TF positive platelets and microvesicles.Therapeutic strategies targeting, beyond systemic inflammation (i.e. with tocilizumab, an anti interleukin-6 receptor), this state of platelet activation might therefore be beneficial. Among the antithrombotic drugs proposed as candidates to treat patients with SARS-CoV-2 infection, antiplatelet drugs, such as aspirin are showing promising results.
  • |Blood Platelets/*metabolism[MESH]
  • |COVID-19/*blood[MESH]
  • |Humans[MESH]


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