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10.3389/fmed.2021.674645 http://scihub22266oqcxt.onion/10.3389/fmed.2021.674645 34368184!8339430!34368184     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=34368184&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Front+Med+(Lausanne) 2021 ; 8 (?): 674645 Nephropedia Template TP {{tp|p=34368184|t=2021. Innate Immune Response to Viral Infections at the Maternal-Fetal Interface in Human Pregnancy |pdf=|usr=}}{{34368184}} gab.com Text Innate Immune Response to Viral Infections at the Maternal-Fetal Interface in Human Pregnancy JO: Front Med (Lausanne) http://www.kidney.de/pget.php?&tw=1&pmid=34368184 #FOAvip The placenta, the first and largest organ to develop after conception, not only nurtures and promotes the development of the conceptus, but, it also functions as a barrier against invading pathogens. Early phases of pregnancy are associated with expansion of specific subsets of Natural Killer cells (dNK) and macrophages (dMphi) at the maternal uterine mucosa, the basal decidua. In concert with cells of fetal origin, dNK cells, and dMphi orchestrate all steps of placenta and fetus development, and provide the first line of defense to limit vertical transmission. However, some pathogens that infect the mother can overcome this protective barrier and jeopardize the fetus health. In this review, we will discuss how members of the classical TORCH family (Toxoplasma, Other, Rubella, Cytomegalovirus, and Herpes simplex virus) and some emerging viruses (Hepatitis E virus, Zika virus, and SARS-CoV2) can afford access to the placental fortress. We will also discuss how changes in the intrauterine environment as a consequence of maternal immune cell activation contribute to placental diseases and devastating pregnancy outcomes. Twit Text FOAVip Innate Immune Response to Viral Infections at the Maternal-Fetal Interface in Human Pregnancy ????Front Med (Lausanne) http://www.kidney.de/pget.php?&tw=1&pmid=34368184 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=34368184 #FOAvip English Wikipedia <ref>{{Cite pmid|34368184}}</ref> Innate Immune Response to Viral Infections at the Maternal-Fetal Interface in Human Pregnancy #MMPMID34368184 Espino A; El Costa H; Tabiasco J; Al-Daccak R; Jabrane-Ferrat N Front Med (Lausanne) 2021[]; 8 (?): 674645 PMID34368184show ga The placenta, the first and largest organ to develop after conception, not only nurtures and promotes the development of the conceptus, but, it also functions as a barrier against invading pathogens. Early phases of pregnancy are associated with expansion of specific subsets of Natural Killer cells (dNK) and macrophages (dMphi) at the maternal uterine mucosa, the basal decidua. In concert with cells of fetal origin, dNK cells, and dMphi orchestrate all steps of placenta and fetus development, and provide the first line of defense to limit vertical transmission. However, some pathogens that infect the mother can overcome this protective barrier and jeopardize the fetus health. In this review, we will discuss how members of the classical TORCH family (Toxoplasma, Other, Rubella, Cytomegalovirus, and Herpes simplex virus) and some emerging viruses (Hepatitis E virus, Zika virus, and SARS-CoV2) can afford access to the placental fortress. We will also discuss how changes in the intrauterine environment as a consequence of maternal immune cell activation contribute to placental diseases and devastating pregnancy outcomes. ?Innate Immune Response to Viral Infections at the Maternal-Fetal Inter(epmc).pdf DeepDyve Pubget Overpricing