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10.1016/j.csbj.2021.07.023 http://scihub22266oqcxt.onion/10.1016/j.csbj.2021.07.023 34336145!8310780!34336145     free     free     free Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Comput+Struct+Biotechnol+J 2021 ; 19 (ä): 4217-4225 Nephropedia Template TP {{tp|p=34336145|t=2021. Immune evasion of SARS-CoV-2 from interferon antiviral system |pdf=|usr=}}{{34336145}} gab.com Text Immune evasion of SARS-CoV-2 from interferon antiviral system JO: Comput Struct Biotechnol J http://www.kidney.de/pget.php?&tw=1&pmid=34336145 #FOAvip The on-going pandemic of coronavirus disease 2019 (COVID-19) caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) has led to unprecedented medical and socioeconomic crises. Although the viral pathogenesis remains elusive, deficiency of effective antiviral interferon (IFN) responses upon SARS-CoV-2 infection has been recognized as a hallmark of COVID-19 contributing to the disease pathology and progress. Recently, multiple proteins encoded by SARS-CoV-2 have been shown to act as potential IFN antagonists with diverse possible mechanisms. Here, we summarize and discuss the strategies of SARS-CoV-2 for evasion of innate immunity (particularly the antiviral IFN responses), understanding of which will facilitate not only the elucidation of SARS-CoV-2 infection and pathogenesis but also the development of antiviral intervention therapies. Twit Text FOAVip Immune evasion of SARS-CoV-2 from interferon antiviral system ????Comput Struct Biotechnol J http://www.kidney.de/pget.php?&tw=1&pmid=34336145 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=34336145 #FOAvip English Wikipedia <ref>{{Cite pmid|34336145}}</ref> Immune evasion of SARS-CoV-2 from interferon antiviral system #MMPMID34336145 Min YQ; Huang M; Sun X; Deng F; Wang H; Ning YJ Comput Struct Biotechnol J 2021[]; 19 (ä): 4217-4225 PMID34336145show ga The on-going pandemic of coronavirus disease 2019 (COVID-19) caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) has led to unprecedented medical and socioeconomic crises. Although the viral pathogenesis remains elusive, deficiency of effective antiviral interferon (IFN) responses upon SARS-CoV-2 infection has been recognized as a hallmark of COVID-19 contributing to the disease pathology and progress. Recently, multiple proteins encoded by SARS-CoV-2 have been shown to act as potential IFN antagonists with diverse possible mechanisms. Here, we summarize and discuss the strategies of SARS-CoV-2 for evasion of innate immunity (particularly the antiviral IFN responses), understanding of which will facilitate not only the elucidation of SARS-CoV-2 infection and pathogenesis but also the development of antiviral intervention therapies. äImmune evasion of SARS-CoV-2 from interferon antiviral system (epmc).pdf DeepDyve Pubget Overpricing