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10.3390/ijms22147538 http://scihub22266oqcxt.onion/10.3390/ijms22147538 34299162!8307949!34299162     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=34299162&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Int+J+Mol+Sci 2021 ; 22 (14): ä Nephropedia Template TP {{tp|p=34299162|t=2021. IgA Vasculitis: Etiology, Treatment, Biomarkers and Epigenetic Changes |pdf=|usr=}}{{34299162}} gab.com Text IgA Vasculitis: Etiology, Treatment, Biomarkers and Epigenetic Changes JO: Int J Mol Sci http://www.kidney.de/pget.php?&tw=1&pmid=34299162 #FOAvip IgA, previously called Henoch-Schonlein vasculitis, is an essential immune component that drives the host immune response to the external environment. As IgA has the unique characteristic of a flexible response to broad types of microorganisms, it sometimes causes an autoreactive response in the host human body. IgA vasculitis and related organ dysfunction are representative IgA-mediated autoimmune diseases; bacterial and viral infections often trigger IgA vasculitis. Recent drug developments and the presence of COVID-19 have revealed that these agents can also trigger IgA vasculitis. These findings provide a novel understanding of the pathogenesis of IgA vasculitis. In this review, we focus on the characteristics of IgA and symptoms of IgA vasculitis and other organ dysfunction. We also mention the therapeutic approach, biomarkers, novel triggers for IgA vasculitis, and epigenetic modifications in patients with IgA vasculitis. Twit Text FOAVip IgA Vasculitis: Etiology, Treatment, Biomarkers and Epigenetic Changes ????Int J Mol Sci http://www.kidney.de/pget.php?&tw=1&pmid=34299162 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=34299162 #FOAvip English Wikipedia <ref>{{Cite pmid|34299162}}</ref> IgA Vasculitis: Etiology, Treatment, Biomarkers and Epigenetic Changes #MMPMID34299162 Sugino H; Sawada Y; Nakamura M Int J Mol Sci 2021[Jul]; 22 (14): ä PMID34299162show ga IgA, previously called Henoch-Schonlein vasculitis, is an essential immune component that drives the host immune response to the external environment. As IgA has the unique characteristic of a flexible response to broad types of microorganisms, it sometimes causes an autoreactive response in the host human body. IgA vasculitis and related organ dysfunction are representative IgA-mediated autoimmune diseases; bacterial and viral infections often trigger IgA vasculitis. Recent drug developments and the presence of COVID-19 have revealed that these agents can also trigger IgA vasculitis. These findings provide a novel understanding of the pathogenesis of IgA vasculitis. In this review, we focus on the characteristics of IgA and symptoms of IgA vasculitis and other organ dysfunction. We also mention the therapeutic approach, biomarkers, novel triggers for IgA vasculitis, and epigenetic modifications in patients with IgA vasculitis. |*Epigenesis, Genetic[MESH] |Animals[MESH] |Biomarkers/*analysis[MESH] |Humans[MESH] |Immunoglobulin A/*metabolism[MESH]IgA Vasculitis: Etiology, Treatment, Biomarkers and Epigenetic Changes(epmc).pdf DeepDyve Pubget Overpricing