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10.1016/j.heliyon.2021.e07548 http://scihub22266oqcxt.onion/10.1016/j.heliyon.2021.e07548 34296023!8270732!34296023     free     free     free Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Heliyon 2021 ; 7 (7): e07548 Nephropedia Template TP {{tp|p=34296023|t=2021. Recent advance of ACE2 and microbiota dysfunction in COVID-19 pathogenesis |pdf=|usr=}}{{34296023}} gab.com Text Recent advance of ACE2 and microbiota dysfunction in COVID-19 pathogenesis JO: Heliyon http://www.kidney.de/pget.php?&tw=1&pmid=34296023 #FOAvip Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) caused the coronavirus disease 2019 (COVID-19) and has become the world's most pressing public health threat. Although not as common as respiratory symptoms, a substantial proportion of patients with COVID-19 presented the gastrointestinal symptoms. ACE2, as the receptor of SARS-CoV and SARS-CoV-2, is highly expressed in the epithelia of the epithelium cells in lung and intestine. In addition, ACE2 is essential for the innate immunity, amino acid transportation and the homeostasis of intestinal microecology. The composition of gut microbiota in COVID-19 patients was altered and concordant with inflammatory, which may explain the gastrointestinal symptoms in patients. Here we reviewed and discussed the evolving role for ACE2 and gut microbiota in SARS-CoV-2 infection which might provide innovative approaches to targeting ACE2 and gut microbiota for the COVID-19 therapy. Twit Text FOAVip Recent advance of ACE2 and microbiota dysfunction in COVID-19 pathogenesis ????Heliyon http://www.kidney.de/pget.php?&tw=1&pmid=34296023 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=34296023 #FOAvip English Wikipedia <ref>{{Cite pmid|34296023}}</ref> Recent advance of ACE2 and microbiota dysfunction in COVID-19 pathogenesis #MMPMID34296023 Yu Z; Yang Z; Wang Y; Zhou F; Li S; Li C; Li L; Zhang W; Li X Heliyon 2021[Jul]; 7 (7): e07548 PMID34296023show ga Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) caused the coronavirus disease 2019 (COVID-19) and has become the world's most pressing public health threat. Although not as common as respiratory symptoms, a substantial proportion of patients with COVID-19 presented the gastrointestinal symptoms. ACE2, as the receptor of SARS-CoV and SARS-CoV-2, is highly expressed in the epithelia of the epithelium cells in lung and intestine. In addition, ACE2 is essential for the innate immunity, amino acid transportation and the homeostasis of intestinal microecology. The composition of gut microbiota in COVID-19 patients was altered and concordant with inflammatory, which may explain the gastrointestinal symptoms in patients. Here we reviewed and discussed the evolving role for ACE2 and gut microbiota in SARS-CoV-2 infection which might provide innovative approaches to targeting ACE2 and gut microbiota for the COVID-19 therapy. äRecent advance of ACE2 and microbiota dysfunction in COVID-19 pathogen(epmc).pdf DeepDyve Pubget Overpricing