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10.3389/fendo.2021.708333

http://scihub22266oqcxt.onion/10.3389/fendo.2021.708333
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suck abstract from ncbi

pmid34276567      Front+Endocrinol+(Lausanne) 2021 ; 12 (?): 708333
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  • Coronavirus Disease 2019 and the Thyroid - Progress and Perspectives #MMPMID34276567
  • Inaba H; Aizawa T
  • Front Endocrinol (Lausanne) 2021[]; 12 (?): 708333 PMID34276567show ga
  • SARS-CoV-2 infection (COVID-19) is currently a tremendous global health problem. COVID-19 causes considerable damage to a wide range of vital organs most prominently the respiratory system. Recently, clinical evidence for thyroidal insults during and after COVID-19 has been accumulated. As of today, almost all non-neoplastic thyroid diseases, i.e., Graves' disease, Hashimoto's thyroiditis, subacute, painless and postpartum thyroiditis, have been reported as a complication of COVID-19, and causality by the virus has been strongly implicated in all of them. Similar thyroid problems have been reported in the past with the SARS-CoV outbreak in 2002. In this review, we briefly look back at the reported evidence of alteration in thyroid functionality and thyroid diseases associated with SARS-CoV and then proceed to examine the issue with COVID-19 in detail, which is then followed by an in-depth discussion regarding a pathogenetic link between Coronavirus infection and thyroid disease.
  • |COVID-19/*complications/*physiopathology[MESH]
  • |Humans[MESH]
  • |Thyroid Diseases/*virology[MESH]


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