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10.1016/j.celrep.2021.109415 http://scihub22266oqcxt.onion/10.1016/j.celrep.2021.109415 34270919!8238662!34270919     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=34270919&cmd=llinks): Failed to open stream: HTTP request failed! 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SARS-CoV-2 variant B 1 617 is resistant to bamlanivimab and evades antibodies induced by infection and vaccination |pdf=|usr=}}{{34270919}} gab.com Text SARS-CoV-2 variant B 1 617 is resistant to bamlanivimab and evades antibodies induced by infection and vaccination JO: Cell Rep http://www.kidney.de/pget.php?&tw=1&pmid=34270919 #FOAvip The emergence of severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) variants threatens efforts to contain the coronavirus disease 2019 (COVID-19) pandemic. The number of COVID-19 cases and deaths in India has risen steeply, and a SARS-CoV-2 variant, B.1.617, is believed to be responsible for many of these cases. The spike protein of B.1.617 harbors two mutations in the receptor binding domain, which interacts with the angiotensin converting enzyme 2 (ACE2) receptor and constitutes the main target of neutralizing antibodies. Therefore, we analyze whether B.1.617 is more adept in entering cells and/or evades antibody responses. B.1.617 enters two of eight cell lines tested with roughly 50% increased efficiency and is equally inhibited by two entry inhibitors. In contrast, B.1.617 is resistant against bamlanivimab, an antibody used for COVID-19 treatment. B.1.617 evades antibodies induced by infection or vaccination, although less so than the B.1.351 variant. Collectively, our study reveals that antibody evasion of B.1.617 may contribute to the rapid spread of this variant. Twit Text FOAVip SARS-CoV-2 variant B 1 617 is resistant to bamlanivimab and evades antibodies induced by infection and vaccination ????Cell Rep http://www.kidney.de/pget.php?&tw=1&pmid=34270919 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=34270919 #FOAvip English Wikipedia <ref>{{Cite pmid|34270919}}</ref> SARS-CoV-2 variant B 1 617 is resistant to bamlanivimab and evades antibodies induced by infection and vaccination #MMPMID34270919 Hoffmann M; Hofmann-Winkler H; Kruger N; Kempf A; Nehlmeier I; Graichen L; Arora P; Sidarovich A; Moldenhauer AS; Winkler MS; Schulz S; Jack HM; Stankov MV; Behrens GMN; Pohlmann S Cell Rep 2021[Jul]; 36 (3): 109415 PMID34270919show ga The emergence of severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) variants threatens efforts to contain the coronavirus disease 2019 (COVID-19) pandemic. The number of COVID-19 cases and deaths in India has risen steeply, and a SARS-CoV-2 variant, B.1.617, is believed to be responsible for many of these cases. The spike protein of B.1.617 harbors two mutations in the receptor binding domain, which interacts with the angiotensin converting enzyme 2 (ACE2) receptor and constitutes the main target of neutralizing antibodies. Therefore, we analyze whether B.1.617 is more adept in entering cells and/or evades antibody responses. B.1.617 enters two of eight cell lines tested with roughly 50% increased efficiency and is equally inhibited by two entry inhibitors. In contrast, B.1.617 is resistant against bamlanivimab, an antibody used for COVID-19 treatment. B.1.617 evades antibodies induced by infection or vaccination, although less so than the B.1.351 variant. Collectively, our study reveals that antibody evasion of B.1.617 may contribute to the rapid spread of this variant. |*COVID-19 Drug Treatment[MESH] |Angiotensin-Converting Enzyme 2/*pharmacology[MESH] |Antibodies, Monoclonal, Humanized/immunology/*pharmacology[MESH] |Antibodies, Viral/immunology/*pharmacology[MESH] |COVID-19/immunology[MESH] |Cell Line[MESH] |Esters/*pharmacology[MESH] |Guanidines/*pharmacology[MESH] |Humans[MESH] |Protease Inhibitors/pharmacology[MESH] |Protein Binding[MESH] |SARS-CoV-2/genetics/*immunology/pathogenicity[MESH] |Spike Glycoprotein, Coronavirus/chemistry/genetics/*immunology[MESH]SARS-CoV-2 variant B 1 617 is resistant to bamlanivimab and evades ant(epmc).pdf DeepDyve Pubget Overpricing