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10.3389/fncel.2021.673217 http://scihub22266oqcxt.onion/10.3389/fncel.2021.673217 34248502!8267916!34248502     free     free     free Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Front+Cell+Neurosci 2021 ; 15 (ä): 673217 Nephropedia Template TP {{tp|p=34248502|t=2021. Endothelial Senescence and Chronic Fatigue Syndrome, a COVID-19 Based Hypothesis |pdf=|usr=}}{{34248502}} gab.com Text Endothelial Senescence and Chronic Fatigue Syndrome, a COVID-19 Based Hypothesis JO: Front Cell Neurosci http://www.kidney.de/pget.php?&tw=1&pmid=34248502 #FOAvip Myalgic encephalomyelitis/chronic fatigue syndrome is a serious illness of unknown etiology, characterized by debilitating exhaustion, memory impairment, pain and sleep abnormalities. Viral infections are believed to initiate the pathogenesis of this syndrome although the definite proof remains elusive. With the unfolding of COVID-19 pandemic, the interest in this condition has resurfaced as excessive tiredness, a major complaint of patients infected with the SARS-CoV-2 virus, often lingers for a long time, resulting in disability, and poor life quality. In a previous article, we hypothesized that COVID-19-upregulated angiotensin II triggered premature endothelial cell senescence, disrupting the intestinal and blood brain barriers. Here, we hypothesize further that post-viral sequelae, including myalgic encephalomyelitis/chronic fatigue syndrome, are promoted by the gut microbes or toxin translocation from the gastrointestinal tract into other tissues, including the brain. This model is supported by the SARS-CoV-2 interaction with host proteins and bacterial lipopolysaccharide. Conversely, targeting microbial translocation and cellular senescence may ameliorate the symptoms of this disabling illness. Twit Text FOAVip Endothelial Senescence and Chronic Fatigue Syndrome, a COVID-19 Based Hypothesis ????Front Cell Neurosci http://www.kidney.de/pget.php?&tw=1&pmid=34248502 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=34248502 #FOAvip English Wikipedia <ref>{{Cite pmid|34248502}}</ref> Endothelial Senescence and Chronic Fatigue Syndrome, a COVID-19 Based Hypothesis #MMPMID34248502 Sfera A; Osorio C; Zapata Martin Del Campo CM; Pereida S; Maurer S; Maldonado JC; Kozlakidis Z Front Cell Neurosci 2021[]; 15 (ä): 673217 PMID34248502show ga Myalgic encephalomyelitis/chronic fatigue syndrome is a serious illness of unknown etiology, characterized by debilitating exhaustion, memory impairment, pain and sleep abnormalities. Viral infections are believed to initiate the pathogenesis of this syndrome although the definite proof remains elusive. With the unfolding of COVID-19 pandemic, the interest in this condition has resurfaced as excessive tiredness, a major complaint of patients infected with the SARS-CoV-2 virus, often lingers for a long time, resulting in disability, and poor life quality. In a previous article, we hypothesized that COVID-19-upregulated angiotensin II triggered premature endothelial cell senescence, disrupting the intestinal and blood brain barriers. Here, we hypothesize further that post-viral sequelae, including myalgic encephalomyelitis/chronic fatigue syndrome, are promoted by the gut microbes or toxin translocation from the gastrointestinal tract into other tissues, including the brain. This model is supported by the SARS-CoV-2 interaction with host proteins and bacterial lipopolysaccharide. Conversely, targeting microbial translocation and cellular senescence may ameliorate the symptoms of this disabling illness. äEndothelial Senescence and Chronic Fatigue Syndrome, a COVID-19 Based (epmc).pdf DeepDyve Pubget Overpricing