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10.1002/acn3.51416

http://scihub22266oqcxt.onion/10.1002/acn3.51416
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34247452!8351384!34247452
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suck abstract from ncbi


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pmid34247452      Ann+Clin+Transl+Neurol 2021 ; 8 (8): 1745-1749
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  • Diaphragm dysfunction in severe COVID-19 as determined by neuromuscular ultrasound #MMPMID34247452
  • Farr E; Wolfe AR; Deshmukh S; Rydberg L; Soriano R; Walter JM; Boon AJ; Wolfe LF; Franz CK
  • Ann Clin Transl Neurol 2021[Aug]; 8 (8): 1745-1749 PMID34247452show ga
  • Many survivors from severe coronavirus disease 2019 (COVID-19) suffer from persistent dyspnea and fatigue long after resolution of the active infection. In a cohort of 21 consecutive severe post-COVID-19 survivors admitted to an inpatient rehabilitation hospital, 16 (76%) of them had at least one sonographic abnormality of diaphragm muscle structure or function. This corresponded to a significant reduction in diaphragm muscle contractility as represented by thickening ratio (muscle thickness at maximal inspiration/end-expiration) for the post-COVID-19 compared to non-COVID-19 cohorts. These findings may shed new light on neuromuscular respiratory dysfunction as a contributor to prolonged functional impairments after hospitalization for post-COVID-19.
  • |*Diaphragm/diagnostic imaging/pathology/physiopathology[MESH]
  • |Adult[MESH]
  • |Aged[MESH]
  • |Aged, 80 and over[MESH]
  • |COVID-19/*complications/diagnostic imaging/pathology/physiopathology[MESH]
  • |Female[MESH]
  • |Hospitals, Rehabilitation[MESH]
  • |Humans[MESH]
  • |Inpatients[MESH]
  • |Male[MESH]
  • |Middle Aged[MESH]
  • |Post-Acute COVID-19 Syndrome[MESH]


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