Use my Search Websuite to scan PubMed, PMCentral, Journal Hosts and Journal Archives, FullText. Kick-your-searchterm to multiple Engines kick-your-query now !>

A dictionary by aggregated review articles of nephrology, medicine and the life sciences Your one-stop-run pathway from word to the immediate pdf of peer-reviewed on-topic knowledge.

10.1016/j.humimm.2021.06.011 http://scihub22266oqcxt.onion/10.1016/j.humimm.2021.06.011 34229864!8245343!34229864     free     free     free Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Hum+Immunol 2021 ; 82 (10): 733-745 Nephropedia Template TP {{tp|p=34229864|t=2021. Humoral immune mechanisms involved in protective and pathological immunity during COVID-19 |pdf=|usr=}}{{34229864}} gab.com Text Humoral immune mechanisms involved in protective and pathological immunity during COVID-19 JO: Hum Immunol http://www.kidney.de/pget.php?&tw=1&pmid=34229864 #FOAvip The severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) causing COVID-19 is associated with excessive inflammation, as a main reason for severe condition and death. Increased inflammatory cytokines and humoral response to SARS-CoV-2 correlate with COVID-19 immunity and pathogenesis. Importantly, the levels of pro-inflammatory cytokines that increase profoundly in systemic circulation appear as part of the clinical pictures of two overlapping conditions, sepsis and the hemophagocytic syndromes. Both conditions can develop lethal inflammatory responses that lead to tissue damage, however, in many patients hemophagocytic lymphohistiocytosis (HLH) can be differentiated from sepsis. This is a key issue because the life-saving aggressive immunosuppressive treatment, required in the HLH therapy, is absent in sepsis guidelines. This paper aims to describe the pathophysiology and clinical relevance of these distinct entities in the course of COVID-19 that resemble sepsis and further highlights two effector arms of the humoral immune response (inflammatory cytokine and immunoglobulin production) during COVID-19 infection. Twit Text FOAVip Humoral immune mechanisms involved in protective and pathological immunity during COVID-19 ????Hum Immunol http://www.kidney.de/pget.php?&tw=1&pmid=34229864 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=34229864 #FOAvip English Wikipedia <ref>{{Cite pmid|34229864}}</ref> Humoral immune mechanisms involved in protective and pathological immunity during COVID-19 #MMPMID34229864 Widjaja G; Turki Jalil A; Sulaiman Rahman H; Abdelbasset WK; Bokov DO; Suksatan W; Ghaebi M; Marofi F; Gholizadeh Navashenaq J; Jadidi-Niaragh F; Ahmadi M Hum Immunol 2021[Oct]; 82 (10): 733-745 PMID34229864show ga The severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) causing COVID-19 is associated with excessive inflammation, as a main reason for severe condition and death. Increased inflammatory cytokines and humoral response to SARS-CoV-2 correlate with COVID-19 immunity and pathogenesis. Importantly, the levels of pro-inflammatory cytokines that increase profoundly in systemic circulation appear as part of the clinical pictures of two overlapping conditions, sepsis and the hemophagocytic syndromes. Both conditions can develop lethal inflammatory responses that lead to tissue damage, however, in many patients hemophagocytic lymphohistiocytosis (HLH) can be differentiated from sepsis. This is a key issue because the life-saving aggressive immunosuppressive treatment, required in the HLH therapy, is absent in sepsis guidelines. This paper aims to describe the pathophysiology and clinical relevance of these distinct entities in the course of COVID-19 that resemble sepsis and further highlights two effector arms of the humoral immune response (inflammatory cytokine and immunoglobulin production) during COVID-19 infection. |Animals[MESH] |COVID-19/*immunology[MESH] |Cytokines/immunology[MESH] |Humans[MESH] |Immunity, Humoral/*immunology[MESH] |Inflammation/immunology[MESH] |Lymphohistiocytosis, Hemophagocytic/immunology[MESH] |SARS-CoV-2/immunology[MESH]Humoral immune mechanisms involved in protective and pathological immu(epmc).pdf DeepDyve Pubget Overpricing