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10.1186/s13054-021-03659-6 http://scihub22266oqcxt.onion/10.1186/s13054-021-03659-6 34229735!8259088!34229735     free     free     free       Crit+Care 2021 ; 25 (1): 236 Nephropedia Template TP {{tp|p=34229735|t=2021. Infectious disease-associated encephalopathies |pdf=|usr=}}{{34229735}} gab.com Text Infectious disease-associated encephalopathies JO: Crit Care http://www.kidney.de/pget.php?&tw=1&pmid=34229735 #FOAvip Infectious diseases may affect brain function and cause encephalopathy even when the pathogen does not directly infect the central nervous system, known as infectious disease-associated encephalopathy. The systemic inflammatory process may result in neuroinflammation, with glial cell activation and increased levels of cytokines, reduced neurotrophic factors, blood-brain barrier dysfunction, neurotransmitter metabolism imbalances, and neurotoxicity, and behavioral and cognitive impairments often occur in the late course. Even though infectious disease-associated encephalopathies may cause devastating neurologic and cognitive deficits, the concept of infectious disease-associated encephalopathies is still under-investigated; knowledge of the underlying mechanisms, which may be distinct from those of encephalopathies of non-infectious cause, is still limited. In this review, we focus on the pathophysiology of encephalopathies associated with peripheral (sepsis, malaria, influenza, and COVID-19), emerging therapeutic strategies, and the role of neuroinflammation. Twit Text FOAVip Infectious disease-associated encephalopathies ????Crit Care http://www.kidney.de/pget.php?&tw=1&pmid=34229735 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=34229735 #FOAvip English Wikipedia <ref>{{Cite pmid|34229735}}</ref> Infectious disease-associated encephalopathies #MMPMID34229735 Barbosa-Silva MC; Lima MN; Battaglini D; Robba C; Pelosi P; Rocco PRM; Maron-Gutierrez T Crit Care 2021[Jul]; 25 (1): 236 PMID34229735show ga Infectious diseases may affect brain function and cause encephalopathy even when the pathogen does not directly infect the central nervous system, known as infectious disease-associated encephalopathy. The systemic inflammatory process may result in neuroinflammation, with glial cell activation and increased levels of cytokines, reduced neurotrophic factors, blood-brain barrier dysfunction, neurotransmitter metabolism imbalances, and neurotoxicity, and behavioral and cognitive impairments often occur in the late course. Even though infectious disease-associated encephalopathies may cause devastating neurologic and cognitive deficits, the concept of infectious disease-associated encephalopathies is still under-investigated; knowledge of the underlying mechanisms, which may be distinct from those of encephalopathies of non-infectious cause, is still limited. In this review, we focus on the pathophysiology of encephalopathies associated with peripheral (sepsis, malaria, influenza, and COVID-19), emerging therapeutic strategies, and the role of neuroinflammation. |Blood-Brain Barrier/immunology[MESH] |Brain Diseases/*immunology/prevention & control[MESH] |COVID-19/*complications/immunology[MESH] |Cytokines/*immunology[MESH] |Humans[MESH] |Influenza, Human/*complications/immunology[MESH] |Malaria/*complications/immunology[MESH]Infectious disease-associated encephalopathies (epmc).pdf DeepDyve Pubget Overpricing