Use my Search Websuite to scan PubMed, PMCentral, Journal Hosts and Journal Archives, FullText. Kick-your-searchterm to multiple Engines kick-your-query now !>

A dictionary by aggregated review articles of nephrology, medicine and the life sciences Your one-stop-run pathway from word to the immediate pdf of peer-reviewed on-topic knowledge.

10.1016/j.scr.2021.102436 http://scihub22266oqcxt.onion/10.1016/j.scr.2021.102436 34186311!8236004!34186311     free     free     free Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Stem+Cell+Res 2021 ; 54 (ä): 102436 Nephropedia Template TP {{tp|p=34186311|t=2021. Non-permissive SARS-CoV-2 infection in human neurospheres |pdf=|usr=}}{{34186311}} gab.com Text Non-permissive SARS-CoV-2 infection in human neurospheres JO: Stem Cell Res http://www.kidney.de/pget.php?&tw=1&pmid=34186311 #FOAvip Coronavirus disease 2019 (COVID-19) was initially described as a viral infection of the respiratory tract. It is now known, however, that several other organs are affected, including the brain. Neurological manifestations such as stroke, encephalitis, and psychiatric conditions have been reported in COVID-19 patients, but the neurotropic potential of the virus is still debated. Herein, we sought to investigate SARS-CoV-2 infection in human neural cells. We demonstrated that SARS-CoV-2 infection of neural tissue is non-permissive, however, it can elicit inflammatory response and cell damage. These findings add to the hypothesis that most of the neural damage caused by SARS-CoV-2 infection is due to a systemic inflammation leading to indirect harmful effects on the central nervous system despite the absence of local viral replication. Twit Text FOAVip Non-permissive SARS-CoV-2 infection in human neurospheres ????Stem Cell Res http://www.kidney.de/pget.php?&tw=1&pmid=34186311 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=34186311 #FOAvip English Wikipedia <ref>{{Cite pmid|34186311}}</ref> Non-permissive SARS-CoV-2 infection in human neurospheres #MMPMID34186311 Pedrosa CDSG; Goto-Silva L; Temerozo JR; Souza LRQ; Vitoria G; Ornelas IM; Karmirian K; Mendes MA; Gomes IC; Sacramento CQ; Fintelman-Rodrigues N; Cardoso Soares V; Silva Gomes Dias SD; Salerno JA; Puig-Pijuan T; Oliveira JT; Aragao LGHS; Torquato TCQ; Verissimo C; Biagi D; Cruvinel EM; Dariolli R; Furtado DR; Borges HL; Bozza PT; Rehen S; Moreno L Souza T; Guimaraes MZP Stem Cell Res 2021[Jul]; 54 (ä): 102436 PMID34186311show ga Coronavirus disease 2019 (COVID-19) was initially described as a viral infection of the respiratory tract. It is now known, however, that several other organs are affected, including the brain. Neurological manifestations such as stroke, encephalitis, and psychiatric conditions have been reported in COVID-19 patients, but the neurotropic potential of the virus is still debated. Herein, we sought to investigate SARS-CoV-2 infection in human neural cells. We demonstrated that SARS-CoV-2 infection of neural tissue is non-permissive, however, it can elicit inflammatory response and cell damage. These findings add to the hypothesis that most of the neural damage caused by SARS-CoV-2 infection is due to a systemic inflammation leading to indirect harmful effects on the central nervous system despite the absence of local viral replication. |*COVID-19[MESH] |*SARS-CoV-2[MESH] |Brain[MESH] |Humans[MESH]Non-permissive SARS-CoV-2 infection in human neurospheres (epmc).pdf DeepDyve Pubget Overpricing