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10.1007/s10554-021-02317-w

http://scihub22266oqcxt.onion/10.1007/s10554-021-02317-w
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34160722!8220422!34160722
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suck abstract from ncbi

pmid34160722      Int+J+Cardiovasc+Imaging 2021 ; 37 (11): 3279-3283
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  • Transient left ventricular clot in COVID-19-related myocarditis is associated with hypereosinophilic syndrome: a case report #MMPMID34160722
  • Ziaie N; Maleh PA; Ramandi MMA; Pourkia R; Latifi K; Mansouri D
  • Int J Cardiovasc Imaging 2021[Nov]; 37 (11): 3279-3283 PMID34160722show ga
  • Frequent clinical presentations have been reported in patients with Coronavirus disease 2019 (COVID-19). It may be associated with multi-organ and cardiovascular involvements such as myocarditis and clot formation. Hypereosinophilic syndrome (HES) is a rare disease diagnosed with idiopathic eosinophilia and organ involvement. Here, we report a patient with COVID-19 who presented with clot formation and myocarditis. One month after discharge, regarding persistent peripheral/bone marrow hypereosinophilia and clot in echocardiography, fluorescent in situ hybridization (FISH) analysis was done that showed FIP1L1-CHIC2 fusion (PDGFRa rearrangement) in 18% of scored cells and PDGFRbeta rearrangement in 12% of scored cells, which confirmed HES diagnosis. Clot formation may be a late manifestation of COVID-19 or myocarditis due to COVID-19, or the first manifestation of HES that COVID-19 might provoke in this rare syndrome.
  • |*COVID-19[MESH]
  • |*Hypereosinophilic Syndrome/complications/diagnostic imaging/drug therapy[MESH]
  • |*Myocarditis/diagnostic imaging/etiology[MESH]
  • |Humans[MESH]
  • |In Situ Hybridization, Fluorescence[MESH]
  • |Oncogene Proteins, Fusion/genetics[MESH]
  • |Predictive Value of Tests[MESH]


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