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10.3389/fimmu.2021.651545 http://scihub22266oqcxt.onion/10.3389/fimmu.2021.651545 34149692!8207198!34149692     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Front+Immunol 2021 ; 12 (ä): 651545 Nephropedia Template TP {{tp|p=34149692|t=2021. Immunothrombosis in Acute Respiratory Dysfunction of COVID-19 |pdf=|usr=}}{{34149692}} gab.com Text Immunothrombosis in Acute Respiratory Dysfunction of COVID-19 JO: Front Immunol http://www.kidney.de/pget.php?&tw=1&pmid=34149692 #FOAvip COVID-19 is an acute, complex disorder that was caused by a new beta-coronavirus severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2). Based on current reports, it was surprising that the characteristics of many patients with COVID-19, who fulfil the Berlin criteria for acute respiratory distress syndrome (ARDS), are not always like those of patients with typical ARDS and can change over time. While the mechanisms of COVID-19-related respiratory dysfunction in COVID-19 have not yet been fully elucidated, pulmonary microvascular thrombosis is speculated to be involved. Considering that thrombosis is highly related to other inflammatory lung diseases, immunothrombosis, a two-way process that links coagulation and inflammation, seems to be involved in the pathophysiology of COVID-19, including respiratory dysfunction. Thus, the current manuscript will describe the proinflammatory milieu in COVID-19, summarize current evidence of thrombosis in COVID-19, and discuss possible interactions between these two. Twit Text FOAVip Immunothrombosis in Acute Respiratory Dysfunction of COVID-19 ????Front Immunol http://www.kidney.de/pget.php?&tw=1&pmid=34149692 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=34149692 #FOAvip English Wikipedia <ref>{{Cite pmid|34149692}}</ref> Immunothrombosis in Acute Respiratory Dysfunction of COVID-19 #MMPMID34149692 Fang XZ; Wang YX; Xu JQ; He YJ; Peng ZK; Shang Y Front Immunol 2021[]; 12 (ä): 651545 PMID34149692show ga COVID-19 is an acute, complex disorder that was caused by a new beta-coronavirus severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2). Based on current reports, it was surprising that the characteristics of many patients with COVID-19, who fulfil the Berlin criteria for acute respiratory distress syndrome (ARDS), are not always like those of patients with typical ARDS and can change over time. While the mechanisms of COVID-19-related respiratory dysfunction in COVID-19 have not yet been fully elucidated, pulmonary microvascular thrombosis is speculated to be involved. Considering that thrombosis is highly related to other inflammatory lung diseases, immunothrombosis, a two-way process that links coagulation and inflammation, seems to be involved in the pathophysiology of COVID-19, including respiratory dysfunction. Thus, the current manuscript will describe the proinflammatory milieu in COVID-19, summarize current evidence of thrombosis in COVID-19, and discuss possible interactions between these two. |COVID-19/*immunology/*pathology[MESH] |Humans[MESH] |Inflammation/immunology/pathology/*virology[MESH] |Respiratory Distress Syndrome/immunology/pathology/*virology[MESH] |SARS-CoV-2[MESH]Immunothrombosis in Acute Respiratory Dysfunction of COVID-19 (epmc).pdf DeepDyve Pubget Overpricing