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10.1093/cvr/cvab200 http://scihub22266oqcxt.onion/10.1093/cvr/cvab200 34114614!8344830!34114614     free     free     free Deprecated: Implicit conversion from float 227.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 227.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 227.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 227.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 227.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 227.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 227.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 227.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 227.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Cardiovasc+Res 2021 ; 117 (10): 2161-2174 Nephropedia Template TP {{tp|p=34114614|t=2021. Progress in cardiac research: from rebooting cardiac regeneration to a complete cell atlas of the heart |pdf=|usr=}}{{34114614}} gab.com Text Progress in cardiac research: from rebooting cardiac regeneration to a complete cell atlas of the heart JO: Cardiovasc Res http://www.kidney.de/pget.php?&tw=1&pmid=34114614 #FOAvip We review some of the important discoveries and advances made in basic and translational cardiac research in 2020. For example, in the field of myocardial infarction (MI), new aspects of autophagy and the importance of eosinophils were described. Novel approaches, such as a glycocalyx mimetic, were used to improve cardiac recovery following MI. The strategy of 3D bio-printing was shown to allow the fabrication of a chambered cardiac organoid. The benefit of combining tissue engineering with paracrine therapy to heal injured myocardium is discussed. We highlight the importance of cell-to-cell communication, in particular, the relevance of extracellular vesicles, such as exosomes, which transport proteins, lipids, non-coding RNAs, and mRNAs and actively contribute to angiogenesis and myocardial regeneration. In this rapidly growing field, new strategies were developed to stimulate the release of reparative exosomes in ischaemic myocardium. Single-cell sequencing technology is causing a revolution in the study of transcriptional expression at cellular resolution, revealing unanticipated heterogeneity within cardiomyocytes, pericytes and fibroblasts, and revealing a unique subpopulation of cardiac fibroblasts. Several studies demonstrated that exosome- and non-coding RNA-mediated approaches can enhance human induced pluripotent stem cell (iPSC) viability and differentiation into mature cardiomyocytes. Important details of the mitochondrial Ca2+ uniporter and its relevance were elucidated. Novel aspects of cancer therapeutic-induced cardiotoxicity were described, such as the novel circular RNA circITCH, which may lead to novel treatments. Finally, we provide some insights into the effects of SARS-CoV-2 on the heart. Twit Text FOAVip Progress in cardiac research: from rebooting cardiac regeneration to a complete cell atlas of the heart ????Cardiovasc Res http://www.kidney.de/pget.php?&tw=1&pmid=34114614 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=34114614 #FOAvip English Wikipedia <ref>{{Cite pmid|34114614}}</ref> Progress in cardiac research: from rebooting cardiac regeneration to a complete cell atlas of the heart #MMPMID34114614 Davidson SM; Padro T; Bollini S; Vilahur G; Duncker DJ; Evans PC; Guzik T; Hoefer IE; Waltenberger J; Wojta J; Weber C Cardiovasc Res 2021[Aug]; 117 (10): 2161-2174 PMID34114614show ga We review some of the important discoveries and advances made in basic and translational cardiac research in 2020. For example, in the field of myocardial infarction (MI), new aspects of autophagy and the importance of eosinophils were described. Novel approaches, such as a glycocalyx mimetic, were used to improve cardiac recovery following MI. The strategy of 3D bio-printing was shown to allow the fabrication of a chambered cardiac organoid. The benefit of combining tissue engineering with paracrine therapy to heal injured myocardium is discussed. We highlight the importance of cell-to-cell communication, in particular, the relevance of extracellular vesicles, such as exosomes, which transport proteins, lipids, non-coding RNAs, and mRNAs and actively contribute to angiogenesis and myocardial regeneration. In this rapidly growing field, new strategies were developed to stimulate the release of reparative exosomes in ischaemic myocardium. Single-cell sequencing technology is causing a revolution in the study of transcriptional expression at cellular resolution, revealing unanticipated heterogeneity within cardiomyocytes, pericytes and fibroblasts, and revealing a unique subpopulation of cardiac fibroblasts. Several studies demonstrated that exosome- and non-coding RNA-mediated approaches can enhance human induced pluripotent stem cell (iPSC) viability and differentiation into mature cardiomyocytes. Important details of the mitochondrial Ca2+ uniporter and its relevance were elucidated. Novel aspects of cancer therapeutic-induced cardiotoxicity were described, such as the novel circular RNA circITCH, which may lead to novel treatments. Finally, we provide some insights into the effects of SARS-CoV-2 on the heart. |*Biomedical Research[MESH] |*Cardiology[MESH] |*Cell Proliferation[MESH] |*Regeneration[MESH] |Animals[MESH] |COVID-19/pathology/virology[MESH] |Cell Communication[MESH] |Cellular Microenvironment[MESH] |Exosomes/metabolism/pathology[MESH] |Heart Failure/metabolism/*pathology/physiopathology[MESH] |Humans[MESH] |Mitochondria, Heart/metabolism/pathology[MESH] |Myocardial Infarction/metabolism/*pathology/physiopathology[MESH] |Myocardial Reperfusion Injury/metabolism/*pathology/physiopathology[MESH] |Myocytes, Cardiac/metabolism/*pathology/virology[MESH] |Phenotype[MESH] |RNA, Untranslated/metabolism[MESH]Progress in cardiac research: from rebooting cardiac regeneration to a(epmc).pdf DeepDyve Pubget Overpricing