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10.3390/biom11050694 http://scihub22266oqcxt.onion/10.3390/biom11050694 34066385!8148218!34066385     free     free     free Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Biomolecules 2021 ; 11 (5): ä Nephropedia Template TP {{tp|p=34066385|t=2021. Immunothrombosis in COVID-19: Implications of Neutrophil Extracellular Traps |pdf=|usr=}}{{34066385}} gab.com Text Immunothrombosis in COVID-19: Implications of Neutrophil Extracellular Traps JO: Biomolecules http://www.kidney.de/pget.php?&tw=1&pmid=34066385 #FOAvip SARS-CoV-2 is a member of the family of coronaviruses associated with severe outbreaks of respiratory diseases in recent decades and is the causative agent of the COVID-19 pandemic. The recognition by and activation of the innate immune response recruits neutrophils, which, through their different mechanisms of action, form extracellular neutrophil traps, playing a role in infection control and trapping viral, bacterial, and fungal etiological agents. However, in patients with COVID-19, activation at the vascular level, combined with other cells and inflammatory mediators, leads to thrombotic events and disseminated intravascular coagulation, thus leading to a series of clinical manifestations in cerebrovascular, cardiac, pulmonary, and kidney disease while promoting severe disease and mortality. Previous studies of hospitalized patients with COVID-19 have shown that elevated levels of markers specific for NETs, such as free DNA, MPO, and H3Cit, are strongly associated with the total neutrophil count; with acute phase reactants that include CRP, D-dimer, lactate dehydrogenase, and interleukin secretion; and with an increased risk of severe COVID-19. This study analyzed the interactions between NETs and the activation pathways involved in immunothrombotic processes in patients with COVID-19. Twit Text FOAVip Immunothrombosis in COVID-19: Implications of Neutrophil Extracellular Traps ????Biomolecules http://www.kidney.de/pget.php?&tw=1&pmid=34066385 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=34066385 #FOAvip English Wikipedia <ref>{{Cite pmid|34066385}}</ref> Immunothrombosis in COVID-19: Implications of Neutrophil Extracellular Traps #MMPMID34066385 Bautista-Becerril B; Campi-Caballero R; Sevilla-Fuentes S; Hernandez-Regino LM; Hanono A; Flores-Bustamante A; Gonzalez-Flores J; Garcia-Avila CA; Aquino-Galvez A; Castillejos-Lopez M; Juarez-Cisneros A; Camarena A Biomolecules 2021[May]; 11 (5): ä PMID34066385show ga SARS-CoV-2 is a member of the family of coronaviruses associated with severe outbreaks of respiratory diseases in recent decades and is the causative agent of the COVID-19 pandemic. The recognition by and activation of the innate immune response recruits neutrophils, which, through their different mechanisms of action, form extracellular neutrophil traps, playing a role in infection control and trapping viral, bacterial, and fungal etiological agents. However, in patients with COVID-19, activation at the vascular level, combined with other cells and inflammatory mediators, leads to thrombotic events and disseminated intravascular coagulation, thus leading to a series of clinical manifestations in cerebrovascular, cardiac, pulmonary, and kidney disease while promoting severe disease and mortality. Previous studies of hospitalized patients with COVID-19 have shown that elevated levels of markers specific for NETs, such as free DNA, MPO, and H3Cit, are strongly associated with the total neutrophil count; with acute phase reactants that include CRP, D-dimer, lactate dehydrogenase, and interleukin secretion; and with an increased risk of severe COVID-19. This study analyzed the interactions between NETs and the activation pathways involved in immunothrombotic processes in patients with COVID-19. |Biomarkers/metabolism[MESH] |COVID-19/immunology/*pathology/virology[MESH] |Complement System Proteins/metabolism[MESH] |Cytokine Release Syndrome/etiology/pathology[MESH] |Disseminated Intravascular Coagulation/etiology/pathology[MESH] |Extracellular Traps/*metabolism[MESH] |Humans[MESH] |Neutrophils/cytology/immunology/metabolism[MESH] |SARS-CoV-2/isolation & purification[MESH]Immunothrombosis in COVID-19: Implications of Neutrophil Extracellular(epmc).pdf DeepDyve Pubget Overpricing