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10.1016/j.bbcan.2021.188564 http://scihub22266oqcxt.onion/10.1016/j.bbcan.2021.188564 33974950!?!33974950 Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=33974950&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Biochim+Biophys+Acta+Rev+Cancer 2021 ; 1876 (1): 188564 Nephropedia Template TP {{tp|p=33974950|t=2021. Epithelial-mesenchymal transition: When tumor cells meet myeloid-derived suppressor cells |pdf=|usr=}}{{33974950}} gab.com Text Epithelial-mesenchymal transition: When tumor cells meet myeloid-derived suppressor cells JO: Biochim Biophys Acta Rev Cancer http://www.kidney.de/pget.php?&tw=1&pmid=33974950 #FOAvip Myeloid-derived suppressor cells (MDSCs) are a heterogeneous myeloid cell population characterized by protumoral functions in the tumor immune network. An increasing number of studies have focused on the biological functions of MDSCs in tumor immunity. Epithelial-mesenchymal transition (EMT) is a cellular plasticity process accompanied by a loss of epithelial phenotypes and an acquisition of mesenchymal phenotypes. In general, tumor cells that undergo EMT are more likely to invade and metastasize. Recently, extensive evidence suggests that EMT is closely related to a highly immunosuppressive environment. This review will summarize the immunosuppressive capacities of MDSC subsets and their distinct role in tumor EMT and further discuss immunotherapy for tumor EMT by targeting MDSCs. Twit Text FOAVip Epithelial-mesenchymal transition: When tumor cells meet myeloid-derived suppressor cells ????Biochim Biophys Acta Rev Cancer http://www.kidney.de/pget.php?&tw=1&pmid=33974950 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33974950 #FOAvip English Wikipedia <ref>{{Cite pmid|33974950}}</ref> Epithelial-mesenchymal transition: When tumor cells meet myeloid-derived suppressor cells #MMPMID33974950 Cai J; Cui Y; Yang J; Wang S Biochim Biophys Acta Rev Cancer 2021[Aug]; 1876 (1): 188564 PMID33974950show ga Myeloid-derived suppressor cells (MDSCs) are a heterogeneous myeloid cell population characterized by protumoral functions in the tumor immune network. An increasing number of studies have focused on the biological functions of MDSCs in tumor immunity. Epithelial-mesenchymal transition (EMT) is a cellular plasticity process accompanied by a loss of epithelial phenotypes and an acquisition of mesenchymal phenotypes. In general, tumor cells that undergo EMT are more likely to invade and metastasize. Recently, extensive evidence suggests that EMT is closely related to a highly immunosuppressive environment. This review will summarize the immunosuppressive capacities of MDSC subsets and their distinct role in tumor EMT and further discuss immunotherapy for tumor EMT by targeting MDSCs. |*Cell Plasticity/drug effects[MESH] |*Epithelial-Mesenchymal Transition/drug effects[MESH] |*Tumor Escape/drug effects[MESH] |*Tumor Microenvironment[MESH] |Animals[MESH] |Antineoplastic Combined Chemotherapy Protocols/therapeutic use[MESH] |Humans[MESH] |Immunotherapy[MESH] |Molecular Targeted Therapy[MESH] |Myeloid-Derived Suppressor Cells/drug effects/*immunology/metabolism[MESH] |Neoplasms/drug therapy/*immunology/metabolism/pathology[MESH]Epithelial-mesenchymal transition: When tumor cells meet myeloid-deriv(epmc).pdf DeepDyve Pubget Overpricing