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10.1016/j.chaos.2021.110952 http://scihub22266oqcxt.onion/10.1016/j.chaos.2021.110952 33967407!8086832!33967407     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Chaos+Solitons+Fractals 2021 ; 147 (ä): 110952 Nephropedia Template TP {{tp|p=33967407|t=2021. A fractional-order differential equation model of COVID-19 infection of epithelial cells |pdf=|usr=}}{{33967407}} gab.com Text A fractional-order differential equation model of COVID-19 infection of epithelial cells JO: Chaos Solitons Fractals http://www.kidney.de/pget.php?&tw=1&pmid=33967407 #FOAvip A novel coronavirus disease (COVID-19) appeared in Wuhan, China in December 2019 and spread around the world at a rapid pace, taking the form of pandemic. There was an urgent need to look for the remedy and control this deadly disease. A new strain of coronavirus called Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) was considered to be responsible for COVID-19. Novel coronavirus (SARS-CoV-2) belongs to the family of coronaviruses crowned with homotrimeric class 1 fusion spike protein (or S protein) on their surfaces. COVID-19 attacks primarily at our throat and lungs epithelial cells. In COVID-19, a stronger adaptive immune response against SARS-CoV-2 can lead to longer recovery time and leads to several complications. In this paper, we propose a mathematical model for examining the consequence of adaptive immune responses to the viral mutation to control disease transmission. We consider three populations, namely, the uninfected epithelial cells, infected cells, and the SARS-CoV-2 virus. We also take into account combination drug therapy on the dynamics of COVID-19 and its effect. We present a fractional-order model representing COVID-19/SARS-CoV-2 infection of epithelial cells. The main aim of our study is to explore the effect of adaptive immune response using fractional order operator to monitor the influence of memory on the cell-biological aspects. Also, we have studied the outcome of an antiviral drug on the system to obstruct the contact between epithelial cells and SARS-CoV-2 to restrict the COVID-19 disease. Numerical simulations have been done to illustrate our analytical findings. Twit Text FOAVip A fractional-order differential equation model of COVID-19 infection of epithelial cells ????Chaos Solitons Fractals http://www.kidney.de/pget.php?&tw=1&pmid=33967407 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33967407 #FOAvip English Wikipedia <ref>{{Cite pmid|33967407}}</ref> A fractional-order differential equation model of COVID-19 infection of epithelial cells #MMPMID33967407 Chatterjee AN; Ahmad B Chaos Solitons Fractals 2021[Jun]; 147 (ä): 110952 PMID33967407show ga A novel coronavirus disease (COVID-19) appeared in Wuhan, China in December 2019 and spread around the world at a rapid pace, taking the form of pandemic. There was an urgent need to look for the remedy and control this deadly disease. A new strain of coronavirus called Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) was considered to be responsible for COVID-19. Novel coronavirus (SARS-CoV-2) belongs to the family of coronaviruses crowned with homotrimeric class 1 fusion spike protein (or S protein) on their surfaces. COVID-19 attacks primarily at our throat and lungs epithelial cells. In COVID-19, a stronger adaptive immune response against SARS-CoV-2 can lead to longer recovery time and leads to several complications. In this paper, we propose a mathematical model for examining the consequence of adaptive immune responses to the viral mutation to control disease transmission. We consider three populations, namely, the uninfected epithelial cells, infected cells, and the SARS-CoV-2 virus. We also take into account combination drug therapy on the dynamics of COVID-19 and its effect. We present a fractional-order model representing COVID-19/SARS-CoV-2 infection of epithelial cells. The main aim of our study is to explore the effect of adaptive immune response using fractional order operator to monitor the influence of memory on the cell-biological aspects. Also, we have studied the outcome of an antiviral drug on the system to obstruct the contact between epithelial cells and SARS-CoV-2 to restrict the COVID-19 disease. Numerical simulations have been done to illustrate our analytical findings. äA fractional-order differential equation model of COVID-19 infection o(epmc).pdf DeepDyve Pubget Overpricing