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10.1186/s13045-021-01082-6

http://scihub22266oqcxt.onion/10.1186/s13045-021-01082-6
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33926500!8082485!33926500
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suck abstract from ncbi


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pmid33926500      J+Hematol+Oncol 2021 ; 14 (1): 72
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  • SARS-CoV-2 interacts with platelets and megakaryocytes via ACE2-independent mechanism #MMPMID33926500
  • Shen S; Zhang J; Fang Y; Lu S; Wu J; Zheng X; Deng F
  • J Hematol Oncol 2021[Apr]; 14 (1): 72 PMID33926500show ga
  • Evidence suggests that platelets may directly interact with SARS-CoV-2, raising the concern whether ACE2 receptor plays a role in this interaction. The current study showed that SARS-CoV-2 interacts with both platelets and megakaryocytes despite the limited efficiency. Abundance of the conventional receptor ACE2 and alternative receptors or co-factors for SARS-CoV-2 entry was characterized in platelets from COVID-19 patients and healthy persons as well as human megakaryocytes based on laboratory tests or previously reported RNA-seq data. The results suggest that SARS-CoV-2 interacts with platelets and megakaryocytes via ACE2-independent mechanism and may regulate alternative receptor expression associated with COVID-19 coagulation dysfunction.
  • |*Virus Attachment[MESH]
  • |Angiotensin-Converting Enzyme 2/analysis[MESH]
  • |Blood Platelets/metabolism/*virology[MESH]
  • |COVID-19/*blood/complications/genetics[MESH]
  • |Cell Line[MESH]
  • |Humans[MESH]
  • |Megakaryocytes/metabolism/*virology[MESH]
  • |Organ Specificity[MESH]
  • |Platelet Activation[MESH]
  • |RNA, Messenger/biosynthesis/genetics[MESH]
  • |RNA, Viral/isolation & purification[MESH]
  • |Receptors, Virus/*physiology[MESH]
  • |SARS-CoV-2/isolation & purification/*physiology[MESH]
  • |Thrombophilia/etiology/virology[MESH]
  • |Transcription, Genetic[MESH]


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