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10.3389/fcimb.2021.655896 http://scihub22266oqcxt.onion/10.3389/fcimb.2021.655896 33889552!8057809!33889552     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Front+Cell+Infect+Microbiol 2021 ; 11 (ä): 655896 Nephropedia Template TP {{tp|p=33889552|t=2021. IgA Antibodies and IgA Deficiency in SARS-CoV-2 Infection |pdf=|usr=}}{{33889552}} gab.com Text IgA Antibodies and IgA Deficiency in SARS-CoV-2 Infection JO: Front Cell Infect Microbiol http://www.kidney.de/pget.php?&tw=1&pmid=33889552 #FOAvip A large repertoire of IgA is produced by B lymphocytes with T-independent and T-dependent mechanisms useful in defense against pathogenic microorganisms and to reduce immune activation. IgA is active against several pathogens, including rotavirus, poliovirus, influenza virus, and SARS-CoV-2. It protects the epithelial barriers from pathogens and modulates excessive immune responses in inflammatory diseases. An early SARS-CoV-2 specific humoral response is dominated by IgA antibodies responses greatly contributing to virus neutralization. The lack of anti-SARS-Cov-2 IgA and secretory IgA (sIgA) might represent a possible cause of COVID-19 severity, vaccine failure, and possible cause of prolonged viral shedding in patients with Primary Antibody Deficiencies, including patients with Selective IgA Deficiency. Differently from other primary antibody deficiency entities, Selective IgA Deficiency occurs in the vast majority of patients as an asymptomatic condition, and it is often an unrecognized, Studies are needed to clarify the open questions raised by possible consequences of a lack of an IgA response to SARS-CoV-2. Twit Text FOAVip IgA Antibodies and IgA Deficiency in SARS-CoV-2 Infection ????Front Cell Infect Microbiol http://www.kidney.de/pget.php?&tw=1&pmid=33889552 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33889552 #FOAvip English Wikipedia <ref>{{Cite pmid|33889552}}</ref> IgA Antibodies and IgA Deficiency in SARS-CoV-2 Infection #MMPMID33889552 Quinti I; Mortari EP; Fernandez Salinas A; Milito C; Carsetti R Front Cell Infect Microbiol 2021[]; 11 (ä): 655896 PMID33889552show ga A large repertoire of IgA is produced by B lymphocytes with T-independent and T-dependent mechanisms useful in defense against pathogenic microorganisms and to reduce immune activation. IgA is active against several pathogens, including rotavirus, poliovirus, influenza virus, and SARS-CoV-2. It protects the epithelial barriers from pathogens and modulates excessive immune responses in inflammatory diseases. An early SARS-CoV-2 specific humoral response is dominated by IgA antibodies responses greatly contributing to virus neutralization. The lack of anti-SARS-Cov-2 IgA and secretory IgA (sIgA) might represent a possible cause of COVID-19 severity, vaccine failure, and possible cause of prolonged viral shedding in patients with Primary Antibody Deficiencies, including patients with Selective IgA Deficiency. Differently from other primary antibody deficiency entities, Selective IgA Deficiency occurs in the vast majority of patients as an asymptomatic condition, and it is often an unrecognized, Studies are needed to clarify the open questions raised by possible consequences of a lack of an IgA response to SARS-CoV-2. |*IgA Deficiency[MESH] |Antibodies, Neutralizing/blood[MESH] |Antibodies, Viral/*blood[MESH] |COVID-19/*immunology[MESH] |Humans[MESH] |Immunoglobulin A/*blood[MESH] |SARS-CoV-2/immunology[MESH]IgA Antibodies and IgA Deficiency in SARS-CoV-2 Infection (epmc).pdf DeepDyve Pubget Overpricing