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10.1080/19336896.2021.1910468 http://scihub22266oqcxt.onion/10.1080/19336896.2021.1910468 33876719!8078534!33876719     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Prion 2021 ; 15 (1): 53-55 Nephropedia Template TP {{tp|p=33876719|t=2021. Hypothesis: AA amyloidosis is a factor causing systemic complications after coronavirus disease |pdf=|usr=}}{{33876719}} gab.com Text Hypothesis: AA amyloidosis is a factor causing systemic complications after coronavirus disease JO: Prion http://www.kidney.de/pget.php?&tw=1&pmid=33876719 #FOAvip The severe course of COVID-19 causes systemic chronic inflammation and thrombosis in a wide variety of organs and tissues. The nature of these inflammations remains a mystery, although they are known to occur against the background of a high level of cytokine production. The high level of cytokines provokes overproduction of the Serum amyloid A (SAA) protein. Moreover, the number of studies has shown that the severe COVID-19 causes SAA overproduction. The authors of these works regard a high level of SAA exclusively as a biomarker of COVID-19. However, it should be borne in mind that overproduction of SAA can cause systemic AA amyloidosis. SAA forms cytotoxic amyloid deposits in various organs and induces inflammation and thrombosis. The consequences of COVID-19 infection are surprisingly similar to the clinical picture that is observed in AA amyloidosis. Here I present the hypothesis that AA amyloidosis is a factor causing systemic complications after coronavirus disease. Twit Text FOAVip Hypothesis: AA amyloidosis is a factor causing systemic complications after coronavirus disease ????Prion http://www.kidney.de/pget.php?&tw=1&pmid=33876719 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33876719 #FOAvip English Wikipedia <ref>{{Cite pmid|33876719}}</ref> Hypothesis: AA amyloidosis is a factor causing systemic complications after coronavirus disease #MMPMID33876719 Galkin AP Prion 2021[Dec]; 15 (1): 53-55 PMID33876719show ga The severe course of COVID-19 causes systemic chronic inflammation and thrombosis in a wide variety of organs and tissues. The nature of these inflammations remains a mystery, although they are known to occur against the background of a high level of cytokine production. The high level of cytokines provokes overproduction of the Serum amyloid A (SAA) protein. Moreover, the number of studies has shown that the severe COVID-19 causes SAA overproduction. The authors of these works regard a high level of SAA exclusively as a biomarker of COVID-19. However, it should be borne in mind that overproduction of SAA can cause systemic AA amyloidosis. SAA forms cytotoxic amyloid deposits in various organs and induces inflammation and thrombosis. The consequences of COVID-19 infection are surprisingly similar to the clinical picture that is observed in AA amyloidosis. Here I present the hypothesis that AA amyloidosis is a factor causing systemic complications after coronavirus disease. |*Amyloidosis/blood/physiopathology/virology[MESH] |*COVID-19/complications/physiopathology[MESH] |*Serum Amyloid A Protein[MESH] |Biomarkers[MESH] |Humans[MESH]Hypothesis: AA amyloidosis is a factor causing systemic complications (epmc).pdf DeepDyve Pubget Overpricing