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10.1097/RHU.0000000000001733 http://scihub22266oqcxt.onion/10.1097/RHU.0000000000001733 33843779!ä!33843779 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Clin+Rheumatol 2022 ; 28 (1): e263-e269 Nephropedia Template TP {{tp|p=33843779|t=2022. Antibodies: Friends, Foes, or Both? Lessons From COVID-19 for the Rheumatologist |pdf=|usr=}}{{33843779}} gab.com Text Antibodies: Friends, Foes, or Both Lessons From COVID-19 for the Rheumatologist JO: J Clin Rheumatol http://www.kidney.de/pget.php?&tw=1&pmid=33843779 #FOAvip Antibodies are a fundamental tool to fight infections but are intrinsically built as a double-edged sword. One side recognizes the microbial antigen, and the other gives a call to arms to fight infection by recruiting immune cells and triggering inflammation. A balanced immune response must combine a potent neutralizing antibody and a swift disposal of the invading agent by innate immune cells with the least tissue damage possible. The longer the immune system takes to control the infection, the higher the possibility for a self-sustaining inflammatory process with potentially fatal consequences for the host. In addition to quantity, the quality of antibodies also matters, because posttranslational modifications altering the N-glycan composition in Fc fractions may help tilt the balance to the effector side, by modifying their affinity for Fc receptors in immune cells. The COVID-19 pandemic has provided a wealth of data bolstering our understanding of the rules governing the production of protective and nonprotective antibodies. Also, it has broadened our understanding of the role of viruses in triggering autoimmunity and inflammation, and widened our knowledge of the different mechanisms that can be activated by viral infection and lead to autoantibody production, inflammation, and progressive tissue damage. In addition, the COVID-19 infection has contributed a great deal to our comprehension of the role of antibodies in the causation of cytokine storms and systemic inflammatory response syndrome, also seen in patients with systemic autoimmune diseases. Twit Text FOAVip Antibodies: Friends, Foes, or Both Lessons From COVID-19 for the Rheumatologist ????J Clin Rheumatol http://www.kidney.de/pget.php?&tw=1&pmid=33843779 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33843779 #FOAvip English Wikipedia <ref>{{Cite pmid|33843779}}</ref> Antibodies: Friends, Foes, or Both? Lessons From COVID-19 for the Rheumatologist #MMPMID33843779 Rodriguez MA; Fuentes-Silva YJ; Vasquez G J Clin Rheumatol 2022[Jan]; 28 (1): e263-e269 PMID33843779show ga Antibodies are a fundamental tool to fight infections but are intrinsically built as a double-edged sword. One side recognizes the microbial antigen, and the other gives a call to arms to fight infection by recruiting immune cells and triggering inflammation. A balanced immune response must combine a potent neutralizing antibody and a swift disposal of the invading agent by innate immune cells with the least tissue damage possible. The longer the immune system takes to control the infection, the higher the possibility for a self-sustaining inflammatory process with potentially fatal consequences for the host. In addition to quantity, the quality of antibodies also matters, because posttranslational modifications altering the N-glycan composition in Fc fractions may help tilt the balance to the effector side, by modifying their affinity for Fc receptors in immune cells. The COVID-19 pandemic has provided a wealth of data bolstering our understanding of the rules governing the production of protective and nonprotective antibodies. Also, it has broadened our understanding of the role of viruses in triggering autoimmunity and inflammation, and widened our knowledge of the different mechanisms that can be activated by viral infection and lead to autoantibody production, inflammation, and progressive tissue damage. In addition, the COVID-19 infection has contributed a great deal to our comprehension of the role of antibodies in the causation of cytokine storms and systemic inflammatory response syndrome, also seen in patients with systemic autoimmune diseases. |*COVID-19[MESH] |*Rheumatologists[MESH] |Antibodies/blood[MESH] |Humans[MESH] |Pandemics[MESH]Antibodies: Friends, Foes, or Both? Lessons From COVID-19 for the Rheu(epmc).pdf DeepDyve Pubget Overpricing