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10.1177/0963689721993769

http://scihub22266oqcxt.onion/10.1177/0963689721993769
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suck abstract from ncbi


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pmid33840257      Cell+Transplant 2021 ; 30 (ä): 963689721993769
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  • Surviving the Rookie Virus, Severe Acute Respiratory Syndrome Coronavirus 2 (SARS-CoV2): The Immunopathology of a SARS-CoV2 Infection #MMPMID33840257
  • Tsai SF; Lu KY; Chuang HM; Liu CA
  • Cell Transplant 2021[Jan]; 30 (ä): 963689721993769 PMID33840257show ga
  • Until July 29th, the number of confirmed coronavirus (COVID-19) cases worldwide has risen to over 16 million, within which 655 k deaths. Severe acute respiratory syndrome coronavirus 2 (SARS-CoV2) emerges as the 11th global pandemic disease, showing the highest infectivity and lowest infection fatality rate. In this review, we compare the immunopathology among SARS-CoV, Middle East respiratory syndrome coronavirus, and SARS-CoV2. SARS-CoV2 is similar to SARS-CoV; it can cause lymphocytopenia and a rising granulocyte count. Here we point out the human body and concentrated society make for an excellent incubator for virus evolution. Most research energies put into developing the SARS-CoV2 vaccine are trying to block virus infection. Sixty-five percent of severe patients die with multiple organ failure, inflammation, and cytokine storm, which indicates that the patient's immune system maintains functionality. Finding a way to trigger the specific T cell subset and plasmablast in our body is the best shot to get away with SARS-CoV2.
  • |Animals[MESH]
  • |COVID-19/*immunology/pathology[MESH]
  • |Coronavirus Infections/immunology/pathology[MESH]
  • |Coronavirus/immunology[MESH]
  • |Cytokine Release Syndrome/immunology/pathology[MESH]
  • |Humans[MESH]
  • |Inflammation/immunology/pathology[MESH]
  • |SARS-CoV-2/*immunology[MESH]
  • |Severe Acute Respiratory Syndrome/immunology/pathology[MESH]


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