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10.1016/j.etap.2021.103657 http://scihub22266oqcxt.onion/10.1016/j.etap.2021.103657 33838330!8025547!33838330     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=33838330&cmd=llinks): Failed to open stream: HTTP request failed! 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The effect of environmental diesel exhaust pollution on SARS-CoV-2 infection: The mechanism of pulmonary ground glass opacity |pdf=|usr=}}{{33838330}} gab.com Text The effect of environmental diesel exhaust pollution on SARS-CoV-2 infection: The mechanism of pulmonary ground glass opacity JO: Environ Toxicol Pharmacol http://www.kidney.de/pget.php?&tw=1&pmid=33838330 #FOAvip Diesel exhaust particles (DEP) are the major components of atmospheric particulate matter (PM) and chronic exposure is recognized to enhance respiratory system complications. Although the spread of SARS-CoV-2 was found to be associated with the PMs, the mechanism by which exposure to DEP increases the risk of SARS-CoV-2 infection is still under discussion. However, diesel fine PM (dPM) elevate the probability of SARS-CoV-2 infection, as it coincides with the increase in the number of ACE2 receptors. Expression of ACE2 and its colocalized activator, transmembrane protease serine 2 (TMPRSS2) facilitate the entry of SARS-CoV-2 into the alveolar epithelial cells exposed to dPM. Thus, the coexistence of PM and SARS-CoV-2 in the environment augments inflammation and exacerbates lung damage. Increased TGF-beta1 expression due to DEP accompanies the proliferation of the extracellular matrix. In this case, "multifocal ground-glass opacity" (GGO) in a CT scan is an indication of a cytokine storm and severe pneumonia in COVID-19. Twit Text FOAVip The effect of environmental diesel exhaust pollution on SARS-CoV-2 infection: The mechanism of pulmonary ground glass opacity ????Environ Toxicol Pharmacol http://www.kidney.de/pget.php?&tw=1&pmid=33838330 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33838330 #FOAvip English Wikipedia <ref>{{Cite pmid|33838330}}</ref> The effect of environmental diesel exhaust pollution on SARS-CoV-2 infection: The mechanism of pulmonary ground glass opacity #MMPMID33838330 Poyraz BM; Engin ED; Engin AB; Engin A Environ Toxicol Pharmacol 2021[Aug]; 86 (ä): 103657 PMID33838330show ga Diesel exhaust particles (DEP) are the major components of atmospheric particulate matter (PM) and chronic exposure is recognized to enhance respiratory system complications. Although the spread of SARS-CoV-2 was found to be associated with the PMs, the mechanism by which exposure to DEP increases the risk of SARS-CoV-2 infection is still under discussion. However, diesel fine PM (dPM) elevate the probability of SARS-CoV-2 infection, as it coincides with the increase in the number of ACE2 receptors. Expression of ACE2 and its colocalized activator, transmembrane protease serine 2 (TMPRSS2) facilitate the entry of SARS-CoV-2 into the alveolar epithelial cells exposed to dPM. Thus, the coexistence of PM and SARS-CoV-2 in the environment augments inflammation and exacerbates lung damage. Increased TGF-beta1 expression due to DEP accompanies the proliferation of the extracellular matrix. In this case, "multifocal ground-glass opacity" (GGO) in a CT scan is an indication of a cytokine storm and severe pneumonia in COVID-19. |Air Pollution/*adverse effects[MESH] |Angiotensin-Converting Enzyme 2/biosynthesis/genetics[MESH] |COVID-19/*diagnostic imaging/*epidemiology[MESH] |Humans[MESH] |Lung/*diagnostic imaging[MESH] |Particulate Matter[MESH] |Tomography, X-Ray Computed[MESH]The effect of environmental diesel exhaust pollution on SARS-CoV-2 inf(epmc).pdf DeepDyve Pubget Overpricing