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10.1177/20503121211002996 http://scihub22266oqcxt.onion/10.1177/20503121211002996 33815798!7989108!33815798     free     free     free Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       SAGE+Open+Med 2021 ; 9 (ä): 20503121211002996 Nephropedia Template TP {{tp|p=33815798|t=2021. Hypercoagulability in COVID-19: A review of the potential mechanisms underlying clotting disorders |pdf=|usr=}}{{33815798}} gab.com Text Hypercoagulability in COVID-19: A review of the potential mechanisms underlying clotting disorders JO: SAGE Open Med http://www.kidney.de/pget.php?&tw=1&pmid=33815798 #FOAvip Severe acute respiratory syndrome coronavirus-2 has emerged as a new viral pandemic, causing Coronavirus disease 2019 (COVID-19) leading to a wide array of symptoms ranging from asymptomatic to severe respiratory failure. However, coagulation disorders have been found in some patients infected with SARS-CoV-2, leading to either a clotting disorder or hemorrhage. Several mechanisms attempt to explain the mechanism behind the pro-coagulant state seen with COVID-19 patients, including different receptor binding, cytokine storm, and direct viral endothelial damage. SARS-CoV-2 has also been recently found to bind to CLEC4M receptor, a receptor that participates in the clearance of von Willebrand Factor and Factor VIII. The competitive binding of SARS-CoV-2 to CLEC4M could lead to decreased clearance, and therefore a promotion of a pro-coagulative state; however, an experimental study needs to be done to prove such an association. Twit Text FOAVip Hypercoagulability in COVID-19: A review of the potential mechanisms underlying clotting disorders ????SAGE Open Med http://www.kidney.de/pget.php?&tw=1&pmid=33815798 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33815798 #FOAvip English Wikipedia <ref>{{Cite pmid|33815798}}</ref> Hypercoagulability in COVID-19: A review of the potential mechanisms underlying clotting disorders #MMPMID33815798 Alam W SAGE Open Med 2021[]; 9 (ä): 20503121211002996 PMID33815798show ga Severe acute respiratory syndrome coronavirus-2 has emerged as a new viral pandemic, causing Coronavirus disease 2019 (COVID-19) leading to a wide array of symptoms ranging from asymptomatic to severe respiratory failure. However, coagulation disorders have been found in some patients infected with SARS-CoV-2, leading to either a clotting disorder or hemorrhage. Several mechanisms attempt to explain the mechanism behind the pro-coagulant state seen with COVID-19 patients, including different receptor binding, cytokine storm, and direct viral endothelial damage. SARS-CoV-2 has also been recently found to bind to CLEC4M receptor, a receptor that participates in the clearance of von Willebrand Factor and Factor VIII. The competitive binding of SARS-CoV-2 to CLEC4M could lead to decreased clearance, and therefore a promotion of a pro-coagulative state; however, an experimental study needs to be done to prove such an association. äHypercoagulability in COVID-19: A review of the potential mechanisms u(epmc).pdf DeepDyve Pubget Overpricing